RON (MST1R) and HGFL (MST1) Co-Overexpression Supports Breast Tumorigenesis through Autocrine and Paracrine Cellular Crosstalk

por Hunt, Brian G., Jones, Angelle, Lester, Carissa, Davis, James C., Benight, Nancy M., Waltz, Susan E.
Publicado 2022

HGFL-mediated RON signaling supports breast cancer stem cell phenotypes via activation of non-canonical β-catenin signaling

por Ruiz-Torres, Sasha J., Benight, Nancy M., Karns, Rebekah A., Lower, Elyse E., Guan, Jun-Lin, Waltz, Susan E.
Publicado 2017

HGFL supports mammary tumorigenesis by enhancing tumor cell intrinsic survival and influencing macrophage and T-cell responses

por Benight, Nancy M., Wagh, Purnima K., Zinser, Glendon M., Peace, Belinda E., Stuart, William D., Vasiliauskas, Juozas, Pathrose, Peterson, Starnes, Sandra L., Waltz, Susan E.
Publicado 2015

The DEK oncogene promotes cellular proliferation through paracrine Wnt signaling in Ron receptor positive breast cancers

por Privette Vinnedge, Lisa M., Benight, Nancy M., Wagh, Purnima K., Pease, Nicholas A., Nashu, Madison A., Serrano-Lopez, Juana, Adams, Allie K., Cancelas, Jose A., Waltz, Susan E., Wells, Susanne I.
Publicado 2014