Stool is a sensitive and noninvasive source of DNA for monitoring expansion in repeat expansion disease mouse models

por Zhao, Xiaonan, McHugh, Cassandra, Coffey, Sydney R., Jimenez, Diego Antonio, Adams, Elizabeth, Carroll, Jeffrey B., Usdin, Karen
Publicado 2022

Early Detection of Apathetic Phenotypes in Huntington’s Disease Knock-in Mice Using Open Source Tools

por Minnig, Shawn, Bragg, Robert M., Tiwana, Hardeep S., Solem, Wes T., Hovander, William S., Vik, Eva-Mari S., Hamilton, Madeline, Legg, Samuel R. W., Shuttleworth, Dominic D., Coffey, Sydney R., Cantle, Jeffrey P., Carroll, Jeffrey B.
Publicado 2018

Huntingtin loss in hepatocytes is associated with altered metabolism, adhesion, and liver zonation

por Bragg, Robert M, Coffey, Sydney R, Cantle, Jeffrey P, Hu, Shikai, Singh, Sucha, Legg, Samuel RW, McHugh, Cassandra A, Toor, Amreen, Zeitlin, Scott O, Kwak, Seung, Howland, David, Vogt, Thomas F, Monga, Satdarshan P, Carroll, Jeffrey B
Publicado 2023

Huntingtin loss in hepatocytes is associated with altered metabolism, adhesion, and liver zonation

por Bragg, Robert M., Coffey, Sydney R., Cantle, Jeffrey P., Hu, Shikai, Singh, Sucha, Legg, Samuel R.W., McHugh, Cassandra A., Toor, Amreen, Zeitlin, Scott O., Kwak, Seung, Howland, David, Vogt, Thomas F., Monga, Satdarshan P., Carroll, Jeffrey B.
Publicado 2023

Peripheral huntingtin silencing does not ameliorate central signs of disease in the B6.Htt(Q111/+) mouse model of Huntington’s disease

por Coffey, Sydney R., Bragg, Robert M., Minnig, Shawn, Ament, Seth A., Cantle, Jeffrey P., Glickenhaus, Anne, Shelnut, Daniel, Carrillo, José M., Shuttleworth, Dominic D., Rodier, Julie-Anne, Noguchi, Kimihiro, Bennett, C. Frank, Price, Nathan D., Kordasiewicz, Holly B., Carroll, Jeffrey B.
Publicado 2017

Transcriptional regulatory networks underlying gene expression changes in Huntington's disease

por Ament, Seth A, Pearl, Jocelynn R, Cantle, Jeffrey P, Bragg, Robert M, Skene, Peter J, Coffey, Sydney R, Bergey, Dani E, Wheeler, Vanessa C, MacDonald, Marcy E, Baliga, Nitin S, Rosinski, Jim, Hood, Leroy E, Carroll, Jeffrey B, Price, Nathan D
Publicado 2018

Motivational, proteostatic and transcriptional deficits precede synapse loss, gliosis and neurodegeneration in the B6.Htt(Q111/+) model of Huntington’s disease

por Bragg, Robert M., Coffey, Sydney R., Weston, Rory M., Ament, Seth A., Cantle, Jeffrey P., Minnig, Shawn, Funk, Cory C., Shuttleworth, Dominic D., Woods, Emily L., Sullivan, Bonnie R., Jones, Lindsey, Glickenhaus, Anne, Anderson, John S., Anderson, Michael D., Dunnett, Stephen B., Wheeler, Vanessa C., MacDonald, Marcy E., Brooks, Simon P., Price, Nathan D., Carroll, Jeffrey B.
Publicado 2017

Corrigendum: Motivational, proteostatic and transcriptional deficits precede synapse loss, gliosis and neurodegeneration in the B6.Htt(Q111/+) model of Huntington’s disease

por Bragg, Robert M., Coffey, Sydney R., Weston, Rory M., Ament, Seth A., Cantle, Jeffrey P., Minnig, Shawn, Funk, Cory C., Shuttleworth, Dominic D., Woods, Emily L., Sullivan, Bonnie R., Jones, Lindsey, Glickenhaus, Anne, Anderson, John S., Anderson, Michael D., Dunnett, Stephen B., Wheeler, Vanessa C., MacDonald, Marcy E., Brooks, Simon P., Price, Nathan D., Carroll, Jeffrey B.
Publicado 2017