Base excision repair processing of abasic site/single-strand break lesions within clustered damage sites associated with XRCC1 deficiency

por Mourgues, Sophie, Lomax, Martine E., O’Neill, Peter
Publicado 2007

Hierarchy of lesion processing governs the repair, double-strand break formation and mutability of three-lesion clustered DNA damage

por Eccles, Laura J., Lomax, Martine E., O’Neill, Peter
Publicado 2010

Delayed repair of radiation induced clustered DNA damage: Friend or foe?

por Eccles, Laura J., O’Neill, Peter, Lomax, Martine E.
Publicado 2011

Disruption of PARP1 function inhibits base excision repair of a sub-set of DNA lesions

por Reynolds, Pamela, Cooper, Sarah, Lomax, Martine, O'Neill, Peter
Publicado 2015

Processing of thymine glycol in a clustered DNA damage site: mutagenic or cytotoxic

por Bellon, Sophie, Shikazono, Naoya, Cunniffe, Siobhan, Lomax, Martine, O’Neill, Peter
Publicado 2009

Reduced repair capacity of a DNA clustered damage site comprised of 8-oxo-7,8-dihydro-2′-deoxyguanosine and 2-deoxyribonolactone results in an increased mutagenic potential of thes...

por Cunniffe, Siobhan, O’Neill, Peter, Greenberg, Marc M., Lomax, Martine E.
Publicado 2014

Increased mutability and decreased repairability of a three-lesion clustered DNA-damaged site comprised of an AP site and bi-stranded 8-oxoG lesions

por Cunniffe, Siobhan, Walker, Alexandra, Stabler, Robert, O’Neill, Peter, Lomax, Martine E.
Publicado 2014

Efficient cleavage of single and clustered AP site lesions within mono-nucleosome templates by CHO-K1 nuclear extract contrasts with retardation of incision by purified APE1

por Eccles, Laura J., Menoni, Hervé, Angelov, Dimitar, Lomax, Martine E., O’Neill, Peter
Publicado 2015