DMD-10 is dispensable for the initial development of amphid sensory neurons and their survival in mature C. elegans

por Nguyen, Irene, Luth, Eric S.
Publicado 2021

Low-Density Lipoprotein Receptor LRP-2 regulates GLR-1 glutamate receptors and glutamatergic behavior in C. elegans

por Rennich, Bethany J, Luth, Eric S, Hofer, Julia, Juo, Peter
Publicado 2023

N-Alpha-Acetylation of α-Synuclein Increases Its Helical Folding Propensity, GM1 Binding Specificity and Resistance to Aggregation

por Bartels, Tim, Kim, Nora C., Luth, Eric S., Selkoe, Dennis J.
Publicado 2014

Purification of α-Synuclein from Human Brain Reveals an Instability of Endogenous Multimers as the Protein Approaches Purity

por Luth, Eric S., Bartels, Tim, Dettmer, Ulf, Kim, Nora C., Selkoe, Dennis J.
Publicado 2014

The Doublesex/Mab-3 domain transcription factor DMD-10 regulates ASH-dependent behavioral responses

por Durbeck, Julia, Breton, Celine, Suter, Michael, Luth, Eric S., McGehee, Annette M.
Publicado 2021

The AP2 clathrin adaptor protein complex regulates the abundance of GLR-1 glutamate receptors in the ventral nerve cord of Caenorhabditis elegans

por Garafalo, Steven D., Luth, Eric S., Moss, Benjamin J., Monteiro, Michael I., Malkin, Emily, Juo, Peter
Publicado 2015

The Snail transcription factor CES-1 regulates glutamatergic behavior in C. elegans

por Park, Lidia, Luth, Eric S., Jones, Kelsey, Hofer, Julia, Nguyen, Irene, Watters, Katherine E., Juo, Peter
Publicado 2021

VER/VEGF receptors regulate AMPA receptor surface levels and glutamatergic behavior

por Luth, Eric S., Hodul, Molly, Rennich, Bethany J., Riccio, Carmino, Hofer, Julia, Markoja, Kaitlin, Juo, Peter
Publicado 2021

Parkinson-causing α-synuclein missense mutations shift native tetramers to monomers as a mechanism for disease initiation

por Dettmer, Ulf, Newman, Andrew J., Soldner, Frank, Luth, Eric S., Kim, Nora C., von Saucken, Victoria E., Sanderson, John B., Jaenisch, Rudolf, Bartels, Tim, Selkoe, Dennis
Publicado 2015

Corrigendum: Parkinson-causing α-synuclein missense mutations shift native tetramers to monomers as a mechanism for disease initiation

por Dettmer, Ulf, Newman, Andrew J., Soldner, Frank, Luth, Eric S., Kim, Nora C., von Saucken, Victoria E., Sanderson, John B., Jaenisch, Rudolf, Bartels, Tim, Selkoe, Dennis
Publicado 2015