Examining the mechanisms that link β-amyloid and α-synuclein pathologies

por Marsh, Samuel E, Blurton-Jones, Mathew
Publicado 2012

HuCNS-SC Human NSCs Fail to Differentiate, Form Ectopic Clusters, and Provide No Cognitive Benefits in a Transgenic Model of Alzheimer's Disease

por Marsh, Samuel E., Yeung, Stephen T., Torres, Maria, Lau, Lydia, Davis, Joy L., Monuki, Edwin S., Poon, Wayne W., Blurton-Jones, Mathew
Publicado 2017

Human Neural Progenitor Transplantation Rescues Behavior and Reduces α‐Synuclein in a Transgenic Model of Dementia with Lewy Bodies

por Goldberg, Natalie R.S., Marsh, Samuel E., Ochaba, Joseph, Shelley, Brandon C., Davtyan, Hayk, Thompson, Leslie M., Steffan, Joan S., Svendsen, Clive N., Blurton‐Jones, Mathew
Publicado 2017

The neuronal retromer can regulate both neuronal and microglial phenotypes of Alzheimer’s disease

por Qureshi, Yasir H., Berman, Diego E., Marsh, Samuel E., Klein, Ronald L., Patel, Vivek M., Simoes, Sabrina, Kannan, Suvarnambiga, Petsko, Gregory A., Stevens, Beth, Small, Scott A.
Publicado 2022

Exposure of iPSC-derived human microglia to brain substrates enables the generation and manipulation of diverse transcriptional states in vitro

por Dolan, Michael-John, Therrien, Martine, Jereb, Saša, Kamath, Tushar, Gazestani, Vahid, Atkeson, Trevor, Marsh, Samuel E., Goeva, Aleksandrina, Lojek, Neal M., Murphy, Sarah, White, Cassandra M., Joung, Julia, Liu, Bingxu, Limone, Francesco, Eggan, Kevin, Hacohen, Nir, Bernstein, Bradley E., Glass, Christopher K., Leinonen, Ville, Blurton-Jones, Mathew, Zhang, Feng, Epstein, Charles B., Macosko, Evan Z., Stevens, Beth
Publicado 2023