Armet/Manf and Creld2 are components of a specialized ER stress response provoked by inappropriate formation of disulphide bonds: implications for genetic skeletal diseases

por Hartley, Claire L., Edwards, Sarah, Mullan, Lorna, Bell, Peter A., Fresquet, Maryline, Boot-Handford, Raymond P., Briggs, Michael D.
Publicado 2013

Cartilage endoplasmic reticulum stress may influence the onset but not the progression of experimental osteoarthritis

por Kung, Louise H. W., Mullan, Lorna, Soul, Jamie, Wang, Ping, Mori, Kazutoshi, Bateman, John F., Briggs, Michael D., Boot-Handford, Raymond P.
Publicado 2019

XBP1-Independent UPR Pathways Suppress C/EBP-β Mediated Chondrocyte Differentiation in ER-Stress Related Skeletal Disease

por Cameron, Trevor L., Bell, Katrina M., Gresshoff, Irma L., Sampurno, Lisa, Mullan, Lorna, Ermann, Joerg, Glimcher, Laurie H., Boot-Handford, Raymond P., Bateman, John F.
Publicado 2015

Increased intracellular proteolysis reduces disease severity in an ER stress–associated dwarfism

por Mullan, Lorna A., Mularczyk, Ewa J., Kung, Louise H., Forouhan, Mitra, Wragg, Jordan M.A., Goodacre, Royston, Bateman, John F., Swanton, Eileithyia, Briggs, Michael D., Boot-Handford, Raymond P.
Publicado 2017