ATMIN is required for the ATM-mediated signaling and recruitment of 53BP1 to DNA damage sites upon replication stress

por Schmidt, Luisa, Wiedner, Marc, Velimezi, Georgia, Prochazkova, Jana, Owusu, Michel, Bauer, Sabine, Loizou, Joanna I.
Publicado 2014

Validating the concept of mutational signatures with isogenic cell models

por Zou, Xueqing, Owusu, Michel, Harris, Rebecca, Jackson, Stephen P., Loizou, Joanna I., Nik-Zainal, Serena
Publicado 2018

DNA Repair Cofactors ATMIN and NBS1 Are Required to Suppress T Cell Activation

por Prochazkova, Jana, Sakaguchi, Shinya, Owusu, Michel, Mazouzi, Abdelghani, Wiedner, Marc, Velimezi, Georgia, Moder, Martin, Turchinovich, Gleb, Hladik, Anastasiya, Gurnhofer, Elisabeth, Hayday, Adrian, Behrens, Axel, Knapp, Sylvia, Kenner, Lukas, Ellmeier, Wilfried, Loizou, Joanna I.
Publicado 2015

Parallel genome-wide screens identify synthetic viable interactions between the BLM helicase complex and Fanconi anemia

por Moder, Martin, Velimezi, Georgia, Owusu, Michel, Mazouzi, Abdelghani, Wiedner, Marc, Ferreira da Silva, Joana, Robinson-Garcia, Lydia, Schischlik, Fiorella, Slavkovsky, Rastislav, Kralovics, Robert, Schuster, Michael, Bock, Christoph, Ideker, Trey, Jackson, Stephen P., Menche, Jörg, Loizou, Joanna I.
Publicado 2017

Map of synthetic rescue interactions for the Fanconi anemia DNA repair pathway identifies USP48

por Velimezi, Georgia, Robinson-Garcia, Lydia, Muñoz-Martínez, Francisco, Wiegant, Wouter W., Ferreira da Silva, Joana, Owusu, Michel, Moder, Martin, Wiedner, Marc, Rosenthal, Sara Brin, Fisch, Kathleen M., Moffat, Jason, Menche, Jörg, van Attikum, Haico, Jackson, Stephen P., Loizou, Joanna I.
Publicado 2018