Site-specific characterization of endogenous SUMOylation across species and organs

por Hendriks, Ivo A., Lyon, David, Su, Dan, Skotte, Niels H., Daniel, Jeremy A., Jensen, Lars J., Nielsen, Michael L.
Publicado 2018

A Quantitative Method for the Specific Assessment of Caspase-6 Activity in Cell Culture

por Ehrnhoefer, Dagmar E., Skotte, Niels H., Savill, Jane, Nguyen, Yen T. N., Ladha, Safia, Cao, Li-Ping, Dullaghan, Edie, Hayden, Michael R.
Publicado 2011

Characterising the RNA-binding protein atlas of the mammalian brain uncovers RBM5 misregulation in mouse models of Huntington’s disease

por Mullari, Meeli, Fossat, Nicolas, Skotte, Niels H., Asenjo-Martinez, Andrea, Humphreys, David T., Bukh, Jens, Kirkeby, Agnete, Scheel, Troels K. H., Nielsen, Michael L.
Publicado 2023

Rational design of antisense oligonucleotides targeting single nucleotide polymorphisms for potent and allele selective suppression of mutant Huntingtin in the CNS

por Østergaard, Michael E., Southwell, Amber L., Kordasiewicz, Holly, Watt, Andrew T., Skotte, Niels H., Doty, Crystal N., Vaid, Kuljeet, Villanueva, Erika B., Swayze, Eric E., Frank Bennett, C., Hayden, Michael R., Seth, Punit P.
Publicado 2013

Preventing mutant huntingtin proteolysis and intermittent fasting promote autophagy in models of Huntington disease

por Ehrnhoefer, Dagmar E., Martin, Dale D. O., Schmidt, Mandi E., Qiu, Xiaofan, Ladha, Safia, Caron, Nicholas S., Skotte, Niels H., Nguyen, Yen T. N., Vaid, Kuljeet, Southwell, Amber L., Engemann, Sabine, Franciosi, Sonia, Hayden, Michael R.
Publicado 2018

Allele-Specific Suppression of Mutant Huntingtin Using Antisense Oligonucleotides: Providing a Therapeutic Option for All Huntington Disease Patients

por Skotte, Niels H., Southwell, Amber L., Østergaard, Michael E., Carroll, Jeffrey B., Warby, Simon C., Doty, Crystal N., Petoukhov, Eugenia, Vaid, Kuljeet, Kordasiewicz, Holly, Watt, Andrew T., Freier, Susan M., Hung, Gene, Seth, Punit P., Bennett, C. Frank, Swayze, Eric E., Hayden, Michael R.
Publicado 2014

Hippocampal disruptions of synaptic and astrocyte metabolism are primary events of early amyloid pathology in the 5xFAD mouse model of Alzheimer’s disease

por Andersen, Jens V., Skotte, Niels H., Christensen, Sofie K., Polli, Filip S., Shabani, Mohammad, Markussen, Kia H., Haukedal, Henriette, Westi, Emil W., Diaz-delCastillo, Marta, Sun, Ramon C., Kohlmeier, Kristi A., Schousboe, Arne, Gentry, Matthew S., Tanila, Heikki, Freude, Kristine K., Aldana, Blanca I., Mann, Matthias, Waagepetersen, Helle S.
Publicado 2021