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MINNIE and MAXI

Detalles Bibliográficos
Lenguaje:	eng fre
Publicado:	CERN 1972
Acceso en línea:	http://cds.cern.ch/record/1754451

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Mechanism of Maxi-K Channel Activation by Dehydrosoyasaponin-I
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Multiple Facets of Maxi-K(+) Channels: The Heme Connection
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Analysis of Maxi-K alpha subunit splice variants in human myometrium
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Oxygen-glucose deprivation induces ATP release via maxi-anion channels in astrocytes
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Saturation and Microsecond Gating of Current Indicate Depletion-induced Instability of the MaxiK Selectivity Filter
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Immediate vs. delayed endosseous integration of maxi implants: a torque removal animal study
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The ATP-Releasing Maxi-Cl Channel: Its Identity, Molecular Partners, and Physiological/Pathophysiological Implications
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Astaxanthin and Docosahexaenoic Acid Reverse the Toxicity of the Maxi-K (BK) Channel Antagonist Mycotoxin Penitrem A
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The Maxi-K (BK) Channel Antagonist Penitrem A as a Novel Breast Cancer-Targeted Therapeutic
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Membrane-delimited Inhibition of Maxi-K Channel Activity by the Intermediate Conductance Ca(2+)-activated K Channel
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Field Campaign Evaluation of Sensors Lufft GMX500 and MaxiMet WS100 in Peruvian Central Andes
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15th IEEE Computer Society international conference : Micros, minis and maxis : technology thrust vs. user requirements
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Tl(+)-induced μs Gating of Current Indicates Instability of the MaxiK Selectivity Filter as Caused by Ion/Pore Interaction
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Metabolic regulation of the K(ATP) and a maxi-K(V) channel in the insulin-secreting RINm5F cell
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Overexcited MaxiK and K(ATP) channels underlie obstructive jaundice-induced vasoconstrictor hyporeactivity of arterial smooth muscle
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Dihydromyricetin improves vascular hyporesponsiveness in experimental sepsis via attenuating the over-excited MaxiK and K(ATP) channels
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The effect of trichostatin-A and tumor necrosis factor on expression of splice variants of the MaxiK and L-type channels in human myometrium
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Insight-HXMT observations of jet-like corona in a black hole X-ray binary MAXI J1820+070
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Positive Inotropic Effects of ATP Released via the Maxi-Anion Channel in Langendorff-Perfused Mouse Hearts Subjected to Ischemia-Reperfusion
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Optimisation of Milk Protein Top-Down Sequencing Using In-Source Collision-Induced Dissociation in the Maxis Quadrupole Time-of-Flight Mass Spectrometer
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