Cargando…
S100A6 participates in initiation of autoimmune encephalitis and is under epigenetic control
INTRODUCTION: Autoimmune encephalitis (AE) is caused by autoantibodies attacking neuronal cell surface antigens and/or synaptic antigens. We previously demonstrated that S100A6 was hypomethylated in patients with AE and that it promoted B lymphocyte infiltration through the simulated blood–brain bar...
Autores principales: | Lin, Chih‐Hsiang, Li, Sung‐Chou, Lin, Ming‐Hong, Ho, Chen‐Jui, Lu, Yan‐Ting, Lin, Yuyu, Lin, Pei‐Hsien, Tsai, Kuo‐Wang, Tsai, Meng‐Han |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
John Wiley and Sons Inc.
2023
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC10013942/ https://www.ncbi.nlm.nih.gov/pubmed/36748983 http://dx.doi.org/10.1002/brb3.2897 |
Ejemplares similares
-
S100A6 Promotes B Lymphocyte Penetration Through the Blood–Brain Barrier in Autoimmune Encephalitis
por: Tsai, Meng-Han, et al.
Publicado: (2019) -
Validating Prediction Tools for Autoimmune Encephalitis in Adult Taiwanese Patients: A Retrospective Study
por: Lu, Yan-Ting, et al.
Publicado: (2023) -
The Different Clinical Features Between Autoimmune and Infectious Status Epilepticus
por: Lin, Chih-Hsiang, et al.
Publicado: (2019) -
Multiomics analyses identified epigenetic modulation of the S100A gene family in Kawasaki disease and their significant involvement in neutrophil transendothelial migration
por: Huang, Lien-Hung, et al.
Publicado: (2018) -
iTRAQ Proteomics Identified the Potential Biomarkers of Coronary Artery Lesion in Kawasaki Disease and In Vitro Studies Demonstrated That S100A4 Treatment Made HCAECs More Susceptible to Neutrophil Infiltration
por: Weng, Ken-Pen, et al.
Publicado: (2022)