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Hypokalemia-Induced Cardiac Arrest

Renal tubular acidosis (RTA) refers to a group of disorders in which the elimination of hydrogen ions from the kidney or the reabsorption of filtered bicarbonate is impaired, resulting in metabolic acidosis. Hypokalemia is also prominent in different types of RTA. We are presenting an interesting ca...

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Autores principales: Casey, Bradley, Hofstrand, Reese, Patel, Divyang, Bahekar, Amol, Chapa-Rodriguez, Alejandro
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Cureus 2023
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC10023261/
https://www.ncbi.nlm.nih.gov/pubmed/36942187
http://dx.doi.org/10.7759/cureus.35034
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author Casey, Bradley
Hofstrand, Reese
Patel, Divyang
Bahekar, Amol
Chapa-Rodriguez, Alejandro
author_facet Casey, Bradley
Hofstrand, Reese
Patel, Divyang
Bahekar, Amol
Chapa-Rodriguez, Alejandro
author_sort Casey, Bradley
collection PubMed
description Renal tubular acidosis (RTA) refers to a group of disorders in which the elimination of hydrogen ions from the kidney or the reabsorption of filtered bicarbonate is impaired, resulting in metabolic acidosis. Hypokalemia is also prominent in different types of RTA. We are presenting an interesting case about a chronic alcoholic patient who presented to the emergency department and was found to be severely hypokalemic. During her hospital stay, she had multiple cardiac arrests likely secondary to her hypokalemia despite adequate treatment with potassium supplementation. We came to the conclusion of distal RTA in our patient based on hyperchloremic metabolic acidosis, sodium bicarbonate of 10 mmol/L, low potassium, blood urea nitrogen, and creatinine within normal limits, alkaline urine, and a positive urinary anion gap. It is likely that the cause of our patient's underlying type 1 RTA was secondary to her chronic alcohol abuse. Her potassium eventually returned to baseline, and she was discharged.
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spelling pubmed-100232612023-03-19 Hypokalemia-Induced Cardiac Arrest Casey, Bradley Hofstrand, Reese Patel, Divyang Bahekar, Amol Chapa-Rodriguez, Alejandro Cureus Cardiology Renal tubular acidosis (RTA) refers to a group of disorders in which the elimination of hydrogen ions from the kidney or the reabsorption of filtered bicarbonate is impaired, resulting in metabolic acidosis. Hypokalemia is also prominent in different types of RTA. We are presenting an interesting case about a chronic alcoholic patient who presented to the emergency department and was found to be severely hypokalemic. During her hospital stay, she had multiple cardiac arrests likely secondary to her hypokalemia despite adequate treatment with potassium supplementation. We came to the conclusion of distal RTA in our patient based on hyperchloremic metabolic acidosis, sodium bicarbonate of 10 mmol/L, low potassium, blood urea nitrogen, and creatinine within normal limits, alkaline urine, and a positive urinary anion gap. It is likely that the cause of our patient's underlying type 1 RTA was secondary to her chronic alcohol abuse. Her potassium eventually returned to baseline, and she was discharged. Cureus 2023-02-15 /pmc/articles/PMC10023261/ /pubmed/36942187 http://dx.doi.org/10.7759/cureus.35034 Text en Copyright © 2023, Casey et al. https://creativecommons.org/licenses/by/3.0/This is an open access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.
spellingShingle Cardiology
Casey, Bradley
Hofstrand, Reese
Patel, Divyang
Bahekar, Amol
Chapa-Rodriguez, Alejandro
Hypokalemia-Induced Cardiac Arrest
title Hypokalemia-Induced Cardiac Arrest
title_full Hypokalemia-Induced Cardiac Arrest
title_fullStr Hypokalemia-Induced Cardiac Arrest
title_full_unstemmed Hypokalemia-Induced Cardiac Arrest
title_short Hypokalemia-Induced Cardiac Arrest
title_sort hypokalemia-induced cardiac arrest
topic Cardiology
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC10023261/
https://www.ncbi.nlm.nih.gov/pubmed/36942187
http://dx.doi.org/10.7759/cureus.35034
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