BGP-15 Protects against Doxorubicin-Induced Cell Toxicity via Enhanced Mitochondrial Function

Doxorubicin (DOX) is an efficacious and commonly used chemotherapeutic agent. However, its clinical use is limited due to dose-dependent cardiotoxicity. Several mechanisms have been proposed to play a role in DOX-induced cardiotoxicity, such as free radical generation, oxidative stress, mitochondria...

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Detalles Bibliográficos
Autores principales: Gyongyosi, Alexandra, Csaki, Nikolett, Peto, Agota, Szoke, Kitti, Fenyvesi, Ferenc, Bacskay, Ildiko, Lekli, Istvan
Formato: Online Artículo Texto
Lenguaje:English
Publicado: MDPI 2023
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC10049233/
https://www.ncbi.nlm.nih.gov/pubmed/36982341
http://dx.doi.org/10.3390/ijms24065269

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC10049233/
https://www.ncbi.nlm.nih.gov/pubmed/36982341
http://dx.doi.org/10.3390/ijms24065269

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