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BGP-15 Protects against Doxorubicin-Induced Cell Toxicity via Enhanced Mitochondrial Function
Doxorubicin (DOX) is an efficacious and commonly used chemotherapeutic agent. However, its clinical use is limited due to dose-dependent cardiotoxicity. Several mechanisms have been proposed to play a role in DOX-induced cardiotoxicity, such as free radical generation, oxidative stress, mitochondria...
Autores principales: | Gyongyosi, Alexandra, Csaki, Nikolett, Peto, Agota, Szoke, Kitti, Fenyvesi, Ferenc, Bacskay, Ildiko, Lekli, Istvan |
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Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
MDPI
2023
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC10049233/ https://www.ncbi.nlm.nih.gov/pubmed/36982341 http://dx.doi.org/10.3390/ijms24065269 |
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