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P018 Effect of CBT-I on sleep-wake state discrepancy in individuals with co-morbid insomnia and sleep apnoea: A randomised controlled trial

INTRODUCTION: Cognitive behavioural therapy for insomnia (CBT-I) is the recommended treatment for insomnia and improves insomnia symptoms. However, CBT-I effects on sleep-wake state discrepancy have not previously been examined in a sample of individuals with co-morbid insomnia and sleep apnoea (COM...

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Detalles Bibliográficos
Autores principales: Bensen-Boakes, D, Osman, A, Lack, L, Catcheside, P, Antic, N, Smith, S, Chai-Coetzer, C, O'Grady, A, Dunn, N, Robinson, J, McEvoy, D, Sweetman, A
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Oxford University Press 2022
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC10109391/
http://dx.doi.org/10.1093/sleepadvances/zpac029.091
Descripción
Sumario:INTRODUCTION: Cognitive behavioural therapy for insomnia (CBT-I) is the recommended treatment for insomnia and improves insomnia symptoms. However, CBT-I effects on sleep-wake state discrepancy have not previously been examined in a sample of individuals with co-morbid insomnia and sleep apnoea (COMISA). This randomised controlled trial investigated the effect of CBT-I, versus no-treatment control, on sleep-wake state discrepancy in people with COMISA. METHODS: 145 participants had their subjective and objective sleep parameters recorded at pre- and post-treatment via overnight sleep diaries and polysomnography. RESULTS: Pre-treatment, individuals with COMISA significantly (p < 0.05) underestimated their sleep duration (M min = −51.9, SD = 94.1) and sleep efficiency (M % = −9.6, SD = 18.3) and significantly (p < 0.05) overestimated sleep onset latency (M min = 34.5, SD = 86.1). Post-treatment, there were significant reductions in sleep-wake state discrepancy parameters in both groups, but no significant interaction effects to support any differences between CBT-I versus waitlist control groups (all interactions, p > 0.367). DISCUSSION: These findings suggest that treating the insomnia aspect of COMISA through CBT-I first may not be an effective method of reducing sleep-wake state discrepancy. This is possibly due to sleep apnoea being untreated, contributing to frequent awakenings and the discrepancy between perceived and polysomnography derived sleep parameters. Future studies could involve a COMISA group that receives treatment for their sleep apnoea symptoms before CBT-I.