Cargando…
Stimulating β-adrenergic receptors promotes synaptic potentiation by switching CaMKII movement from LTD to LTP mode
Learning, memory, and cognition are thought to require synaptic plasticity, specifically including hippocampal long-term potentiation and depression (LTP and LTD). LTP versus LTD is induced by high-frequency stimulation versus low-frequency, but stimulating β-adrenergic receptors (βARs) enables LTP...
Autores principales: | Larsen, Matthew E., Buonarati, Olivia R., Qian, Hai, Hell, Johannes W., Bayer, K. Ulrich |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
American Society for Biochemistry and Molecular Biology
2023
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC10200978/ https://www.ncbi.nlm.nih.gov/pubmed/37061000 http://dx.doi.org/10.1016/j.jbc.2023.104706 |
Ejemplares similares
-
CaMKII holoenzyme mechanisms that govern the LTP versus LTD decision
por: Cook, Sarah G., et al.
Publicado: (2021) -
Induction and binary expression of LTP/LTD in a minimal model of the CaMKII system
por: Graupner, Michael, et al.
Publicado: (2007) -
GluN2B S1303 phosphorylation by CaMKII or DAPK1: no indication for involvement in ischemia or LTP
por: Tullis, Jonathan E., et al.
Publicado: (2021) -
LTP induction by structural rather than enzymatic functions of CaMKII
por: Tullis, Jonathan E., et al.
Publicado: (2023) -
Ca(2+)- and CaMKII-mediated processes in early LTP
por: Byth, Lachlan A
Publicado: (2014)