BACH1 deficiency prevents neointima formation and maintains the differentiated phenotype of vascular smooth muscle cells by regulating chromatin accessibility

The transcription factor BTB and CNC homology 1(BACH1) has been linked to coronary artery disease risk by human genome-wide association studies, but little is known about the role of BACH1 in vascular smooth muscle cell (VSMC) phenotype switching and neointima formation following vascular injury. Th...

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Detalles Bibliográficos
Autores principales: Guo, Jieyu, Qiu, Jingjing, Jia, Mengping, Li, Qinhan, Wei, Xiangxiang, Li, Liliang, Pan, Qi, Jin, Jiayu, Ge, Fei, Ma, Siyu, He, Yunquan, Lin, Jiayi, Li, Yongbo, Ma, Jinghua, Jiang, Nan, Zhi, Xiuling, Jiang, Lindi, Zhang, Jianyi, Osto, Elena, Jing, Qing, Wang, Xinhong, Meng, Dan
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Oxford University Press 2023
Materias:
Gene regulation, Chromatin and Epigenetics
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC10201429/
https://www.ncbi.nlm.nih.gov/pubmed/36864760
http://dx.doi.org/10.1093/nar/gkad120

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