Cargando…
Defects in Glutathione System in an Animal Model of Amyotrophic Lateral Sclerosis
Amyotrophic lateral sclerosis (ALS) is a progredient neurodegenerative disease characterized by a degeneration of the first and second motor neurons. Elevated levels of reactive oxygen species (ROS) and decreased levels of glutathione, which are important defense mechanisms against ROS, have been re...
Autores principales: | Wunsch, Franziska T., Metzler-Nolte, Nils, Theiss, Carsten, Matschke, Veronika |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
MDPI
2023
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC10215445/ https://www.ncbi.nlm.nih.gov/pubmed/37237880 http://dx.doi.org/10.3390/antiox12051014 |
Ejemplares similares
-
Little Helpers or Mean Rogue—Role of Microglia in Animal Models of Amyotrophic Lateral Sclerosis
por: Cihankaya, Hilal, et al.
Publicado: (2021) -
Deregulated miR-29b-3p Correlates with Tissue-Specific Activation of Intrinsic Apoptosis in An Animal Model of Amyotrophic Lateral Sclerosis
por: Klatt, Christina L., et al.
Publicado: (2019) -
Increased ROS-Dependent Fission of Mitochondria Causes Abnormal Morphology of the Cell Powerhouses in a Murine Model of Amyotrophic Lateral Sclerosis
por: Stein, Jan, et al.
Publicado: (2021) -
Defective cholesterol metabolism in amyotrophic lateral sclerosis
por: Abdel-Khalik, Jonas, et al.
Publicado: (2017) -
Glutathione S-Transferase Rescues Motor Neuronal Toxicity in Fly Model of Amyotrophic Lateral Sclerosis
por: Cha, Sun Joo, et al.
Publicado: (2020)