Cargando…
Oxidization of optic atrophy 1 cysteines occurs during heart ischemia-reperfusion and amplifies cell death by oxidative stress
During cardiac ischemia-reperfusion, excess reactive oxygen species can damage mitochondrial, cellular and organ function. Here we show that cysteine oxidation of the mitochondrial protein Opa1 contributes to mitochondrial damage and cell death caused by oxidative stress. Oxy-proteomics of ischemic-...
Autores principales: | Semenzato, Martina, Kohr, Mark J., Quirin, Charlotte, Menabò, Roberta, Alanova, Petra, Alan, Lukas, Pellattiero, Anna, Murphy, Elizabeth, Di Lisa, Fabio, Scorrano, Luca |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Elsevier
2023
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC10220262/ https://www.ncbi.nlm.nih.gov/pubmed/37224696 http://dx.doi.org/10.1016/j.redox.2023.102755 |
Ejemplares similares
-
Dynamic Regulation of Cysteine Oxidation and Phosphorylation in Myocardial Ischemia–Reperfusion Injury
por: Casin, Kevin M., et al.
Publicado: (2021) -
The cristae modulator Optic atrophy 1 requires mitochondrial ATP synthase oligomers to safeguard mitochondrial function
por: Quintana-Cabrera, Rubén, et al.
Publicado: (2018) -
Oxidative Stress in Intestinal Ischemia-Reperfusion
por: Li, Guangyao, et al.
Publicado: (2022) -
Increased Endogenous Activity of the Renin-Angiotensin System Reduces Infarct Size in the Rats with Early Angiotensin II-dependent Hypertension which Survive the Acute Ischemia/Reperfusion Injury
por: Husková, Zuzana, et al.
Publicado: (2021) -
Oxidation of HMGB1 Causes Attenuation of Its Pro-Inflammatory Activity and Occurs during Liver Ischemia and Reperfusion
por: Liu, Anding, et al.
Publicado: (2012)