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Helicobacter pylori infection selectively attenuates endothelial function in male mice via exosomes-mediated ROS production
BACKGROUND: Substantial sex differences exist in atherosclerosis. Excessive reactive oxygen species (ROS) formation could lead to endothelial dysfunction which is critical to atherosclerosis development and progression. Helicobacter pylori (H. pylori) infection has been shown to attenuate endothelia...
Autores principales: | Zhang, Linfang, Xia, Xiujuan, Wu, Hao, Liu, Xuanyou, Zhu, Qiang, Wang, Meifang, Hao, Hong, Cui, Yuqi, Li, De-Pei, Chen, Shi-You, Martinez-Lemus, Luis A., Hill, Michael A., Xu, Canxia, Liu, Zhenguo |
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Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Frontiers Media S.A.
2023
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC10233065/ https://www.ncbi.nlm.nih.gov/pubmed/37274312 http://dx.doi.org/10.3389/fcimb.2023.1142387 |
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