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A Mycobacterium tuberculosis Effector Targets Mitochondrion, Controls Energy Metabolism, and Limits Cytochrome c Exit

Host metabolism reprogramming is a key feature of Mycobacterium tuberculosis (Mtb) infection that enables the survival of this pathogen within phagocytic cells and modulates the immune response facilitating the spread of the tuberculosis disease. Here, we demonstrate that a previously uncharacterize...

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Detalles Bibliográficos
Autores principales: Martin, Marianne, deVisch, Angelique, Boudehen, Yves-Marie, Barthe, Philippe, Gutierrez, Claude, Turapov, Obolbek, Aydogan, Talip, Heriaud, Laurène, Gracy, Jerome, Neyrolles, Olivier, Mukamolova, Galina V., Letourneur, François, Cohen-Gonsaud, Martin
Formato: Online Artículo Texto
Lenguaje:English
Publicado: American Society for Microbiology 2023
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC10269737/
https://www.ncbi.nlm.nih.gov/pubmed/37036353
http://dx.doi.org/10.1128/spectrum.01066-23

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