Cargando…
Endogenous Amyloid-formed Ca(2+)-permeable Channels in Aged 3xTg AD Mice
Alzheimer’s disease (AD), the leading cause of dementia, is characterized by the accumulation of beta-amyloid peptides (Aβ). However, whether Aβ itself is a key toxic agent in AD pathogenesis and the precise mechanism of Aβ-elicited neurotoxicity are still debated. Emerging evidence demonstrates tha...
Autores principales: | Li, Shuangtao, Ji, Xiaoyu, Gao, Ming, Huang, Bing, Peng, Shuang, Wu, Jie |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Oxford University Press
2023
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC10278988/ https://www.ncbi.nlm.nih.gov/pubmed/37342418 http://dx.doi.org/10.1093/function/zqad025 |
Ejemplares similares
-
The Effects of Physical Running on Dendritic Spines and Amyloid-beta Pathology in 3xTg-AD Male Mice
por: Xu, Benke, et al.
Publicado: (2022) -
Dopaminergic dysfunction in the 3xTg-AD mice model of Alzheimer’s disease
por: Gloria, Yesica, et al.
Publicado: (2021) -
Olfactory dysfunction in the 3xTg-AD model of Alzheimer’s disease
por: Mitrano, Darlene A., et al.
Publicado: (2021) -
Neuroprotective Effects of Probiotic-Supplemented Diet on Cognitive Behavior of 3xTg-AD Mice
por: Tan, Chenxi, et al.
Publicado: (2022) -
Effects of Dietary Supplementation of Carnosine on Mitochondrial Dysfunction, Amyloid Pathology, and Cognitive Deficits in 3xTg-AD Mice
por: Corona, Carlo, et al.
Publicado: (2011)