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Restoration of mitochondrial function by Spirulina polysaccharide via upregulated SOD2 in aging fibroblasts
Reactive oxygen species (ROS), such as superoxide, are crucial factors involved in the stimulation of cellular aging. Mitochondria, which are important organelles responsible for various metabolic processes in cells, produce ROS. These ROS impair mitochondrial function, thereby accelerating aging-re...
Autores principales: | , , , , , , , |
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Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Elsevier
2023
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC10319841/ https://www.ncbi.nlm.nih.gov/pubmed/37416477 http://dx.doi.org/10.1016/j.isci.2023.107113 |
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author | Machihara, Kayo Oki, Shoma Maejima, Yuka Kageyama, Sou Onda, Ayumu Koseki, Yurino Imai, Yasuyuki Namba, Takushi |
author_facet | Machihara, Kayo Oki, Shoma Maejima, Yuka Kageyama, Sou Onda, Ayumu Koseki, Yurino Imai, Yasuyuki Namba, Takushi |
author_sort | Machihara, Kayo |
collection | PubMed |
description | Reactive oxygen species (ROS), such as superoxide, are crucial factors involved in the stimulation of cellular aging. Mitochondria, which are important organelles responsible for various metabolic processes in cells, produce ROS. These ROS impair mitochondrial function, thereby accelerating aging-related cellular dysfunction. Herein, we demonstrated that the Spirulina polysaccharide complex (SPC) restores mitochondrial function and collagen production by scavenging superoxide via the upregulation of superoxide dismutase 2 (SOD2) in aging fibroblasts. We observed that SOD2 expression was linked to inflammatory pathways; however, SPC did not upregulate the expression of most inflammatory cytokines produced as a result of induction of LPS in aging fibroblasts, indicating that SPC induces SOD2 without activation of inflammatory pathways. Furthermore, SPC stimulated endoplasmic reticulum (ER) protein folding by upregulating ER chaperones expression. Thus, SPC is proposed to be an antiaging material that rejuvenates aging fibroblasts by increasing their antioxidant potential via the upregulation of SOD2. |
format | Online Article Text |
id | pubmed-10319841 |
institution | National Center for Biotechnology Information |
language | English |
publishDate | 2023 |
publisher | Elsevier |
record_format | MEDLINE/PubMed |
spelling | pubmed-103198412023-07-06 Restoration of mitochondrial function by Spirulina polysaccharide via upregulated SOD2 in aging fibroblasts Machihara, Kayo Oki, Shoma Maejima, Yuka Kageyama, Sou Onda, Ayumu Koseki, Yurino Imai, Yasuyuki Namba, Takushi iScience Article Reactive oxygen species (ROS), such as superoxide, are crucial factors involved in the stimulation of cellular aging. Mitochondria, which are important organelles responsible for various metabolic processes in cells, produce ROS. These ROS impair mitochondrial function, thereby accelerating aging-related cellular dysfunction. Herein, we demonstrated that the Spirulina polysaccharide complex (SPC) restores mitochondrial function and collagen production by scavenging superoxide via the upregulation of superoxide dismutase 2 (SOD2) in aging fibroblasts. We observed that SOD2 expression was linked to inflammatory pathways; however, SPC did not upregulate the expression of most inflammatory cytokines produced as a result of induction of LPS in aging fibroblasts, indicating that SPC induces SOD2 without activation of inflammatory pathways. Furthermore, SPC stimulated endoplasmic reticulum (ER) protein folding by upregulating ER chaperones expression. Thus, SPC is proposed to be an antiaging material that rejuvenates aging fibroblasts by increasing their antioxidant potential via the upregulation of SOD2. Elsevier 2023-06-14 /pmc/articles/PMC10319841/ /pubmed/37416477 http://dx.doi.org/10.1016/j.isci.2023.107113 Text en © 2023 The Authors https://creativecommons.org/licenses/by/4.0/This is an open access article under the CC BY license (http://creativecommons.org/licenses/by/4.0/). |
spellingShingle | Article Machihara, Kayo Oki, Shoma Maejima, Yuka Kageyama, Sou Onda, Ayumu Koseki, Yurino Imai, Yasuyuki Namba, Takushi Restoration of mitochondrial function by Spirulina polysaccharide via upregulated SOD2 in aging fibroblasts |
title | Restoration of mitochondrial function by Spirulina polysaccharide via upregulated SOD2 in aging fibroblasts |
title_full | Restoration of mitochondrial function by Spirulina polysaccharide via upregulated SOD2 in aging fibroblasts |
title_fullStr | Restoration of mitochondrial function by Spirulina polysaccharide via upregulated SOD2 in aging fibroblasts |
title_full_unstemmed | Restoration of mitochondrial function by Spirulina polysaccharide via upregulated SOD2 in aging fibroblasts |
title_short | Restoration of mitochondrial function by Spirulina polysaccharide via upregulated SOD2 in aging fibroblasts |
title_sort | restoration of mitochondrial function by spirulina polysaccharide via upregulated sod2 in aging fibroblasts |
topic | Article |
url | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC10319841/ https://www.ncbi.nlm.nih.gov/pubmed/37416477 http://dx.doi.org/10.1016/j.isci.2023.107113 |
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