Loss of epidermal glucocorticoid receptor protects against whole body metabolic dysfunction upon chronic corticosterone treatment

OBJECTIVE: Glucocorticoid (GC) excess contributes to the development of metabolic syndrome, defined by visceral obesity, abnormal glucose tolerance, and dyslipidemia. While it is accepted that loss of metabolic control is causative of cutaneous diseases, the systemic effects of epidermal dysfunction...

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Detalles Bibliográficos
Autores principales: Gallego, Andrea, Pérez, Paloma
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Elsevier 2023
Materias:
Brief Communication
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC10345122/
https://www.ncbi.nlm.nih.gov/pubmed/37364709
http://dx.doi.org/10.1016/j.molmet.2023.101763

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC10345122/
https://www.ncbi.nlm.nih.gov/pubmed/37364709
http://dx.doi.org/10.1016/j.molmet.2023.101763

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