Cargando…
Cardiac resident macrophages: key regulatory mediators in the aftermath of myocardial infarction
Acute myocardial infarction (MI) is a prevalent and highly fatal global disease. Despite significant reduction in mortality rates with standard treatment regimens, the risk of heart failure (HF) remains high, necessitating innovative approaches to protect cardiac function and prevent HF progression....
Autores principales: | Chen, Cong, Wang, Jie, Liu, Chao, Hu, Jun |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Frontiers Media S.A.
2023
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC10348646/ https://www.ncbi.nlm.nih.gov/pubmed/37457720 http://dx.doi.org/10.3389/fimmu.2023.1207100 |
Ejemplares similares
-
CTRP1 Aggravates Cardiac Dysfunction Post Myocardial Infarction by Modulating TLR4 in Macrophages
por: Gu, Yang, et al.
Publicado: (2021) -
TRIM21 aggravates cardiac injury after myocardial infarction by promoting M1 macrophage polarization
por: Li, Zhiqiang, et al.
Publicado: (2022) -
Macrophage CARD9 mediates cardiac injury following myocardial infarction through regulation of lipocalin 2 expression
por: Liu, Yan, et al.
Publicado: (2023) -
Self-renewing resident cardiac macrophages limit adverse remodeling following myocardial infarction
por: Dick, Sarah A, et al.
Publicado: (2018) -
Cardiac monocytes and macrophages after myocardial infarction
por: Peet, Claire, et al.
Publicado: (2020)