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Short-term nitisinone discontinuation of hereditary tyrosinemia type 1 mice causes metabolic alterations in glutathione metabolism/biosynthesis and multiple amino acid degradation pathways
| Autores principales: | , , , , , , , , , , , , , |
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| Formato: | Online Artículo Texto |
| Lenguaje: | English |
| Publicado: |
Chongqing Medical University
2022
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| Materias: | |
| Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC10363636/ https://www.ncbi.nlm.nih.gov/pubmed/37492745 http://dx.doi.org/10.1016/j.gendis.2022.11.013 |
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| author | Haaike, Colemonts-Vroninks Brendan, Norman P. Sven, Van Laere Andrew, Davison S. Lionel, Marcélis Georges, Casimir Philippe, Goyens Paul, Claes Dimitri, De Bundel Geert, Martens Rao, Ranganath Lakshminarayan Tamara, Vanhaecke James A, Gallagher Joery, De Kock |
| author_facet | Haaike, Colemonts-Vroninks Brendan, Norman P. Sven, Van Laere Andrew, Davison S. Lionel, Marcélis Georges, Casimir Philippe, Goyens Paul, Claes Dimitri, De Bundel Geert, Martens Rao, Ranganath Lakshminarayan Tamara, Vanhaecke James A, Gallagher Joery, De Kock |
| author_sort | Haaike, Colemonts-Vroninks |
| collection | PubMed |
| description | |
| format | Online Article Text |
| id | pubmed-10363636 |
| institution | National Center for Biotechnology Information |
| language | English |
| publishDate | 2022 |
| publisher | Chongqing Medical University |
| record_format | MEDLINE/PubMed |
| spelling | pubmed-103636362023-07-25 Short-term nitisinone discontinuation of hereditary tyrosinemia type 1 mice causes metabolic alterations in glutathione metabolism/biosynthesis and multiple amino acid degradation pathways Haaike, Colemonts-Vroninks Brendan, Norman P. Sven, Van Laere Andrew, Davison S. Lionel, Marcélis Georges, Casimir Philippe, Goyens Paul, Claes Dimitri, De Bundel Geert, Martens Rao, Ranganath Lakshminarayan Tamara, Vanhaecke James A, Gallagher Joery, De Kock Genes Dis Rapid Communication Chongqing Medical University 2022-12-26 /pmc/articles/PMC10363636/ /pubmed/37492745 http://dx.doi.org/10.1016/j.gendis.2022.11.013 Text en © 2022 The Authors. Publishing services by Elsevier B.V. on behalf of KeAi Communications Co., Ltd. https://creativecommons.org/licenses/by-nc-nd/4.0/This is an open access article under the CC BY-NC-ND license (http://creativecommons.org/licenses/by-nc-nd/4.0/). |
| spellingShingle | Rapid Communication Haaike, Colemonts-Vroninks Brendan, Norman P. Sven, Van Laere Andrew, Davison S. Lionel, Marcélis Georges, Casimir Philippe, Goyens Paul, Claes Dimitri, De Bundel Geert, Martens Rao, Ranganath Lakshminarayan Tamara, Vanhaecke James A, Gallagher Joery, De Kock Short-term nitisinone discontinuation of hereditary tyrosinemia type 1 mice causes metabolic alterations in glutathione metabolism/biosynthesis and multiple amino acid degradation pathways |
| title | Short-term nitisinone discontinuation of hereditary tyrosinemia type 1 mice causes metabolic alterations in glutathione metabolism/biosynthesis and multiple amino acid degradation pathways |
| title_full | Short-term nitisinone discontinuation of hereditary tyrosinemia type 1 mice causes metabolic alterations in glutathione metabolism/biosynthesis and multiple amino acid degradation pathways |
| title_fullStr | Short-term nitisinone discontinuation of hereditary tyrosinemia type 1 mice causes metabolic alterations in glutathione metabolism/biosynthesis and multiple amino acid degradation pathways |
| title_full_unstemmed | Short-term nitisinone discontinuation of hereditary tyrosinemia type 1 mice causes metabolic alterations in glutathione metabolism/biosynthesis and multiple amino acid degradation pathways |
| title_short | Short-term nitisinone discontinuation of hereditary tyrosinemia type 1 mice causes metabolic alterations in glutathione metabolism/biosynthesis and multiple amino acid degradation pathways |
| title_sort | short-term nitisinone discontinuation of hereditary tyrosinemia type 1 mice causes metabolic alterations in glutathione metabolism/biosynthesis and multiple amino acid degradation pathways |
| topic | Rapid Communication |
| url | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC10363636/ https://www.ncbi.nlm.nih.gov/pubmed/37492745 http://dx.doi.org/10.1016/j.gendis.2022.11.013 |
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