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A transient increase of HIF-1α during the G1 phase (G1-HIF) ensures cell survival under nutritional stress
The family of hypoxia-inducible transcription factors (HIF) is activated to adapt cells to low oxygen conditions, but is also known to regulate some biological processes under normoxic conditions. Here we show that HIF-1α protein levels transiently increase during the G1 phase of the cell cycle (des...
Autores principales: | , , , , , , , |
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Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Nature Publishing Group UK
2023
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC10374543/ https://www.ncbi.nlm.nih.gov/pubmed/37500648 http://dx.doi.org/10.1038/s41419-023-06012-7 |
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author | Belapurkar, Ratnal Pfisterer, Maximilian Dreute, Jan Werner, Sebastian Zukunft, Sven Fleming, Ingrid Kracht, Michael SCHMITZ, M. Lienhard |
author_facet | Belapurkar, Ratnal Pfisterer, Maximilian Dreute, Jan Werner, Sebastian Zukunft, Sven Fleming, Ingrid Kracht, Michael SCHMITZ, M. Lienhard |
author_sort | Belapurkar, Ratnal |
collection | PubMed |
description | The family of hypoxia-inducible transcription factors (HIF) is activated to adapt cells to low oxygen conditions, but is also known to regulate some biological processes under normoxic conditions. Here we show that HIF-1α protein levels transiently increase during the G1 phase of the cell cycle (designated as G1-HIF) in an AMP-activated protein kinase (AMPK)-dependent manner. The transient elimination of G1-HIF by a degron system revealed its contribution to cell survival under unfavorable metabolic conditions. Indeed, G1-HIF plays a key role in the cell cycle-dependent expression of genes encoding metabolic regulators and the maintenance of mTOR activity under conditions of nutrient deprivation. Accordingly, transient elimination of G1-HIF led to a significant reduction in the concentration of key proteinogenic amino acids and carbohydrates. These data indicate that G1-HIF acts as a cell cycle-dependent surveillance factor that prevents the onset of starvation-induced apoptosis. |
format | Online Article Text |
id | pubmed-10374543 |
institution | National Center for Biotechnology Information |
language | English |
publishDate | 2023 |
publisher | Nature Publishing Group UK |
record_format | MEDLINE/PubMed |
spelling | pubmed-103745432023-07-29 A transient increase of HIF-1α during the G1 phase (G1-HIF) ensures cell survival under nutritional stress Belapurkar, Ratnal Pfisterer, Maximilian Dreute, Jan Werner, Sebastian Zukunft, Sven Fleming, Ingrid Kracht, Michael SCHMITZ, M. Lienhard Cell Death Dis Article The family of hypoxia-inducible transcription factors (HIF) is activated to adapt cells to low oxygen conditions, but is also known to regulate some biological processes under normoxic conditions. Here we show that HIF-1α protein levels transiently increase during the G1 phase of the cell cycle (designated as G1-HIF) in an AMP-activated protein kinase (AMPK)-dependent manner. The transient elimination of G1-HIF by a degron system revealed its contribution to cell survival under unfavorable metabolic conditions. Indeed, G1-HIF plays a key role in the cell cycle-dependent expression of genes encoding metabolic regulators and the maintenance of mTOR activity under conditions of nutrient deprivation. Accordingly, transient elimination of G1-HIF led to a significant reduction in the concentration of key proteinogenic amino acids and carbohydrates. These data indicate that G1-HIF acts as a cell cycle-dependent surveillance factor that prevents the onset of starvation-induced apoptosis. Nature Publishing Group UK 2023-07-27 /pmc/articles/PMC10374543/ /pubmed/37500648 http://dx.doi.org/10.1038/s41419-023-06012-7 Text en © The Author(s) 2023 https://creativecommons.org/licenses/by/4.0/Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/ (https://creativecommons.org/licenses/by/4.0/) . |
spellingShingle | Article Belapurkar, Ratnal Pfisterer, Maximilian Dreute, Jan Werner, Sebastian Zukunft, Sven Fleming, Ingrid Kracht, Michael SCHMITZ, M. Lienhard A transient increase of HIF-1α during the G1 phase (G1-HIF) ensures cell survival under nutritional stress |
title | A transient increase of HIF-1α during the G1 phase (G1-HIF) ensures cell survival under nutritional stress |
title_full | A transient increase of HIF-1α during the G1 phase (G1-HIF) ensures cell survival under nutritional stress |
title_fullStr | A transient increase of HIF-1α during the G1 phase (G1-HIF) ensures cell survival under nutritional stress |
title_full_unstemmed | A transient increase of HIF-1α during the G1 phase (G1-HIF) ensures cell survival under nutritional stress |
title_short | A transient increase of HIF-1α during the G1 phase (G1-HIF) ensures cell survival under nutritional stress |
title_sort | transient increase of hif-1α during the g1 phase (g1-hif) ensures cell survival under nutritional stress |
topic | Article |
url | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC10374543/ https://www.ncbi.nlm.nih.gov/pubmed/37500648 http://dx.doi.org/10.1038/s41419-023-06012-7 |
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