Cargando…
Post-myocardial infarction treatment with resiniferatoxin modulates the expression of important genes involved in inflammation, plaque stability and angiogenesis
Ventricular tachycardia (VT) and ventricular fibrillation (VF) are the most frequent causes of death in the first 24 hours after myocardial infarction. Previous studies showed that depleting TRPV1 receptors with resiniferatoxin (RTX) led to a reduced risk of VT and VF post-myocardial infarction. The...
Autor principal: | Mircea, Andrei Alexandru |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Applied Systems srl
2023
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC10425609/ https://www.ncbi.nlm.nih.gov/pubmed/37588971 http://dx.doi.org/10.15190/d.2023.2 |
Ejemplares similares
-
Therapeutic Effects of Resiniferatoxin Related with Immunological Responses for Intestinal Inflammation in Trichinellosis
por: Muñoz-Carrillo, José Luis, et al.
Publicado: (2017) -
Perineural resiniferatoxin selectively inhibits inflammatory hyperalgesia
por: Neubert, John K, et al.
Publicado: (2008) -
Thoracic Dorsal Root Ganglion Application of Resiniferatoxin Reduces Myocardial Ischemia-Induced Ventricular Arrhythmias
por: Yamaguchi, Tomoki, et al.
Publicado: (2023) -
Experimental acute myocardial infarction: telocytes involvement in neo-angiogenesis
por: Manole, C G, et al.
Publicado: (2011) -
Resiniferatoxin: The Evolution of the “Molecular Scalpel” for Chronic Pain Relief
por: Brown, Dorothy Cimino
Publicado: (2016)