Cargando…
A Ca(2+) cycling defect connects insulin resistance and heart failure
In a recent study published in Life Metabolism, Quan et al. reported that intracellular Ca(2+) dysregulation in cardiomyocyte can be both a cause and an effect of cardiac insulin resistance that ultimately leads to diabetic cardiomyopathy.
Autores principales: | Shi, Qian, Hall, Duane D., Song, Long-Sheng |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
2022
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC10426325/ https://www.ncbi.nlm.nih.gov/pubmed/37583938 http://dx.doi.org/10.1093/lifemeta/loac014 |
Ejemplares similares
-
Astragalus Granule Prevents Ca(2+) Current Remodeling in Heart Failure by the Downregulation of CaMKII
por: Li, Sinai, et al.
Publicado: (2017) -
Ca(2+) cycling properties are conserved despite bradycardic effects of heart failure in sinoatrial node cells
por: Verkerk, Arie O., et al.
Publicado: (2015) -
Ca(2+) Cycling Impairment in Heart Failure Is Exacerbated by Fibrosis: Insights Gained From Mechanistic Simulations
por: Mora, Maria T., et al.
Publicado: (2018) -
Ca(2+) Cycling in Heart Cells from Ground Squirrels: Adaptive Strategies for Intracellular Ca(2+) Homeostasis
por: Li, Xiao-Chen, et al.
Publicado: (2011) -
Cardiac Insulin Resistance in Heart Failure: The Role of Mitochondrial Dynamics
por: Saotome, Masao, et al.
Publicado: (2019)