Cargando…
Neuroprotection from protein misfolding in cerebral hypoperfusion concurrent with metabolic syndrome. A translational perspective
Based on clinical and experimental evidence, metabolic syndrome (MetS) and type 2 diabetes (T2D) are considered risk factors for chronic cerebral hypoperfusion (CCH) and neurodegeneration. Scientific evidence suggests that protein misfolding is a potential mechanism that explains how CCH can lead to...
Autores principales: | Bordet, Sofía, Luaces, Juan Pablo, Herrera, Maria Ines, Gonzalez, Liliana Mirta, Kobiec, Tamara, Perez-Lloret, Santiago, Otero-Losada, Matilde, Capani, Francisco |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Frontiers Media S.A.
2023
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC10463751/ https://www.ncbi.nlm.nih.gov/pubmed/37650104 http://dx.doi.org/10.3389/fnins.2023.1215041 |
Ejemplares similares
-
Neuroprotection Targeting Protein Misfolding on Chronic Cerebral Hypoperfusion in the Context of Metabolic Syndrome
por: Herrera, María I., et al.
Publicado: (2018) -
The Renin–Angiotensin System Modulates Dopaminergic Neurotransmission: A New Player on the Scene
por: Kobiec, Tamara, et al.
Publicado: (2021) -
Parkinson's Disease in the Era of a Novel Respiratory Virus Pandemic
por: Otero-Losada, Matilde, et al.
Publicado: (2020) -
Neuroprotection in metabolic syndrome by environmental enrichment. A lifespan perspective
por: Kobiec, Tamara, et al.
Publicado: (2023) -
Corrigendum: Neuroprotection in metabolic syndrome by environmental enrichment. A lifespan perspective
por: Kobiec, Tamara, et al.
Publicado: (2023)