Cargando…
Crosstalk between Aldosterone and Glycation through Rac-1 Induces Diabetic Nephropathy
[Image: see text] Background: Advanced glycation end products (AGEs) interaction with its receptor (RAGE) and aldosterone (Aldo) through the mineralocorticoid receptor (MR) activates Rac-1 and NF-κB independently in diabetic nephropathy (DN). However, the crosstalk of Aldo with AGEs-RAGE is still un...
Autores principales: | Apte, Mayura, Khan, Mohd Shahnawaz, Bangar, Nilima, Gvalani, Armaan, Naz, Huma, Tupe, Rashmi S. |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
American Chemical Society
2023
|
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC10568578/ https://www.ncbi.nlm.nih.gov/pubmed/37841153 http://dx.doi.org/10.1021/acsomega.3c05085 |
Ejemplares similares
-
Glycation-Associated Diabetic Nephropathy and the Role of Long Noncoding RNAs
por: Durge, Ankita, et al.
Publicado: (2022) -
Editorial: Myriad types of cell death in nephropathy and their veiled potential
por: Wada, Jun, et al.
Publicado: (2023) -
Attenuation of glycation-induced multiple protein modifications by Indian antidiabetic plant extracts
por: Tupe, Rashmi S., et al.
Publicado: (2016) -
Oxidative damages in tubular epithelial cells in IgA nephropathy: role of crosstalk between angiotensin II and aldosterone
por: Leung, Joseph CK, et al.
Publicado: (2011) -
The role of TGF-β and its crosstalk with RAC1/RAC1b signaling in breast and pancreas carcinoma
por: Melzer, Catharina, et al.
Publicado: (2017)