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Uniparental disomy: expanding the clinical and molecular phenotypes of whole chromosomes
Uniparental disomy (UPD) refers to as both homologous chromosomes inherited from only one parent without identical copies from the other parent. Studies on clinical phenotypes in UPDs are usually focused on the documented UPD 6, 7, 11, 14, 15, and 20, which directly lead to imprinting disorders. Thi...
Autores principales: | , , , , , , , |
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Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Frontiers Media S.A.
2023
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Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC10582337/ https://www.ncbi.nlm.nih.gov/pubmed/37860673 http://dx.doi.org/10.3389/fgene.2023.1232059 |
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author | Chen, Qi Chen, Yunpeng Shi, Lin Tao, Ying Li, Xiaoguang Zhu, Xiaolan Yang, Yan Xu, Wenlin |
author_facet | Chen, Qi Chen, Yunpeng Shi, Lin Tao, Ying Li, Xiaoguang Zhu, Xiaolan Yang, Yan Xu, Wenlin |
author_sort | Chen, Qi |
collection | PubMed |
description | Uniparental disomy (UPD) refers to as both homologous chromosomes inherited from only one parent without identical copies from the other parent. Studies on clinical phenotypes in UPDs are usually focused on the documented UPD 6, 7, 11, 14, 15, and 20, which directly lead to imprinting disorders. This study describes clinical phenotypes and genetic findings of three patients with UPD 2, 9, and 14, respectively. Chromosomal microarray (CMA), UPDtool, methylation-specific multiplex ligation-dependent probe amplification (MS-MLPA) and whole-exome sequencing (WES) analysis were performed to characterize the genetic etiology. The CMA revealed a homozygous region involving the whole chromosome 2 and 9, a partial region of homozygosity in chromosome 14. UPD-tool revealed a paternal origin of the UPD2. MS-MLPA showed hypomethylation of imprinting gene MEG3 from maternal origin in the UPD14 case. In addition, UPD14 case displayed complex symptoms including growth failure, hypotonia and acute respiratory distress syndrome (ARDS), accompanied by several gene mutations with heterozygous genotype by WES analysis. Furthermore, we reviewed the documented UPDs and summarized the clinical characteristics and prognosis. This study highlighted the importance to confirm the diagnosis and origin of UPD using genetic testing. Therefore, it is suggested that expanding of the detailed phenotypes and genotypes provide effective guidance for molecule testing and genetic counseling, and promote further biological investigation to the underlying mechanisms of imprinted disorders and accompanied copy number variations. |
format | Online Article Text |
id | pubmed-10582337 |
institution | National Center for Biotechnology Information |
language | English |
publishDate | 2023 |
publisher | Frontiers Media S.A. |
record_format | MEDLINE/PubMed |
spelling | pubmed-105823372023-10-19 Uniparental disomy: expanding the clinical and molecular phenotypes of whole chromosomes Chen, Qi Chen, Yunpeng Shi, Lin Tao, Ying Li, Xiaoguang Zhu, Xiaolan Yang, Yan Xu, Wenlin Front Genet Genetics Uniparental disomy (UPD) refers to as both homologous chromosomes inherited from only one parent without identical copies from the other parent. Studies on clinical phenotypes in UPDs are usually focused on the documented UPD 6, 7, 11, 14, 15, and 20, which directly lead to imprinting disorders. This study describes clinical phenotypes and genetic findings of three patients with UPD 2, 9, and 14, respectively. Chromosomal microarray (CMA), UPDtool, methylation-specific multiplex ligation-dependent probe amplification (MS-MLPA) and whole-exome sequencing (WES) analysis were performed to characterize the genetic etiology. The CMA revealed a homozygous region involving the whole chromosome 2 and 9, a partial region of homozygosity in chromosome 14. UPD-tool revealed a paternal origin of the UPD2. MS-MLPA showed hypomethylation of imprinting gene MEG3 from maternal origin in the UPD14 case. In addition, UPD14 case displayed complex symptoms including growth failure, hypotonia and acute respiratory distress syndrome (ARDS), accompanied by several gene mutations with heterozygous genotype by WES analysis. Furthermore, we reviewed the documented UPDs and summarized the clinical characteristics and prognosis. This study highlighted the importance to confirm the diagnosis and origin of UPD using genetic testing. Therefore, it is suggested that expanding of the detailed phenotypes and genotypes provide effective guidance for molecule testing and genetic counseling, and promote further biological investigation to the underlying mechanisms of imprinted disorders and accompanied copy number variations. Frontiers Media S.A. 2023-10-04 /pmc/articles/PMC10582337/ /pubmed/37860673 http://dx.doi.org/10.3389/fgene.2023.1232059 Text en Copyright © 2023 Chen, Chen, Shi, Tao, Li, Zhu, Yang and Xu. https://creativecommons.org/licenses/by/4.0/This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms. |
spellingShingle | Genetics Chen, Qi Chen, Yunpeng Shi, Lin Tao, Ying Li, Xiaoguang Zhu, Xiaolan Yang, Yan Xu, Wenlin Uniparental disomy: expanding the clinical and molecular phenotypes of whole chromosomes |
title | Uniparental disomy: expanding the clinical and molecular phenotypes of whole chromosomes |
title_full | Uniparental disomy: expanding the clinical and molecular phenotypes of whole chromosomes |
title_fullStr | Uniparental disomy: expanding the clinical and molecular phenotypes of whole chromosomes |
title_full_unstemmed | Uniparental disomy: expanding the clinical and molecular phenotypes of whole chromosomes |
title_short | Uniparental disomy: expanding the clinical and molecular phenotypes of whole chromosomes |
title_sort | uniparental disomy: expanding the clinical and molecular phenotypes of whole chromosomes |
topic | Genetics |
url | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC10582337/ https://www.ncbi.nlm.nih.gov/pubmed/37860673 http://dx.doi.org/10.3389/fgene.2023.1232059 |
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