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Retracted: Irisin Attenuates Oxidative Stress, Mitochondrial Dysfunction, and Apoptosis in the H9C2 Cellular Model of Septic Cardiomyopathy through Augmenting Fundc1-Dependent Mitophagy

Detalles Bibliográficos
Autor principal: Longevity, Oxidative Medicine and Cellular
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Hindawi 2023
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC10586407/
https://www.ncbi.nlm.nih.gov/pubmed/37868725
http://dx.doi.org/10.1155/2023/9790868
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author Longevity, Oxidative Medicine and Cellular
author_facet Longevity, Oxidative Medicine and Cellular
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spelling pubmed-105864072023-10-20 Retracted: Irisin Attenuates Oxidative Stress, Mitochondrial Dysfunction, and Apoptosis in the H9C2 Cellular Model of Septic Cardiomyopathy through Augmenting Fundc1-Dependent Mitophagy Longevity, Oxidative Medicine and Cellular Oxid Med Cell Longev Retraction Hindawi 2023-10-11 /pmc/articles/PMC10586407/ /pubmed/37868725 http://dx.doi.org/10.1155/2023/9790868 Text en Copyright © 2023 Oxidative Medicine and Cellular Longevity. https://creativecommons.org/licenses/by/4.0/This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.
spellingShingle Retraction
Longevity, Oxidative Medicine and Cellular
Retracted: Irisin Attenuates Oxidative Stress, Mitochondrial Dysfunction, and Apoptosis in the H9C2 Cellular Model of Septic Cardiomyopathy through Augmenting Fundc1-Dependent Mitophagy
title Retracted: Irisin Attenuates Oxidative Stress, Mitochondrial Dysfunction, and Apoptosis in the H9C2 Cellular Model of Septic Cardiomyopathy through Augmenting Fundc1-Dependent Mitophagy
title_full Retracted: Irisin Attenuates Oxidative Stress, Mitochondrial Dysfunction, and Apoptosis in the H9C2 Cellular Model of Septic Cardiomyopathy through Augmenting Fundc1-Dependent Mitophagy
title_fullStr Retracted: Irisin Attenuates Oxidative Stress, Mitochondrial Dysfunction, and Apoptosis in the H9C2 Cellular Model of Septic Cardiomyopathy through Augmenting Fundc1-Dependent Mitophagy
title_full_unstemmed Retracted: Irisin Attenuates Oxidative Stress, Mitochondrial Dysfunction, and Apoptosis in the H9C2 Cellular Model of Septic Cardiomyopathy through Augmenting Fundc1-Dependent Mitophagy
title_short Retracted: Irisin Attenuates Oxidative Stress, Mitochondrial Dysfunction, and Apoptosis in the H9C2 Cellular Model of Septic Cardiomyopathy through Augmenting Fundc1-Dependent Mitophagy
title_sort retracted: irisin attenuates oxidative stress, mitochondrial dysfunction, and apoptosis in the h9c2 cellular model of septic cardiomyopathy through augmenting fundc1-dependent mitophagy
topic Retraction
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC10586407/
https://www.ncbi.nlm.nih.gov/pubmed/37868725
http://dx.doi.org/10.1155/2023/9790868
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