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Correction: NAMPT/SIRT2-mediated inhibition of the p53-p21 signaling pathway is indispensable for maintenance and hematopoietic differentiation of human iPS cells

Detalles Bibliográficos
Autores principales: Xu, Yun, Nasri, Masoud, Dannenmann, Benjamin, Mir, Perihan, Zahabi, Azadeh, Welte, Karl, Morishima, Tatsuya, Skokowa, Julia
Formato: Online Artículo Texto
Lenguaje:English
Publicado: BioMed Central 2023
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC10588213/
https://www.ncbi.nlm.nih.gov/pubmed/37858174
http://dx.doi.org/10.1186/s13287-023-03511-4
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author Xu, Yun
Nasri, Masoud
Dannenmann, Benjamin
Mir, Perihan
Zahabi, Azadeh
Welte, Karl
Morishima, Tatsuya
Skokowa, Julia
author_facet Xu, Yun
Nasri, Masoud
Dannenmann, Benjamin
Mir, Perihan
Zahabi, Azadeh
Welte, Karl
Morishima, Tatsuya
Skokowa, Julia
author_sort Xu, Yun
collection PubMed
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spelling pubmed-105882132023-10-21 Correction: NAMPT/SIRT2-mediated inhibition of the p53-p21 signaling pathway is indispensable for maintenance and hematopoietic differentiation of human iPS cells Xu, Yun Nasri, Masoud Dannenmann, Benjamin Mir, Perihan Zahabi, Azadeh Welte, Karl Morishima, Tatsuya Skokowa, Julia Stem Cell Res Ther Correction BioMed Central 2023-10-20 /pmc/articles/PMC10588213/ /pubmed/37858174 http://dx.doi.org/10.1186/s13287-023-03511-4 Text en © The Author(s) 2023 https://creativecommons.org/licenses/by/4.0/Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/ (https://creativecommons.org/licenses/by/4.0/) . The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/ (https://creativecommons.org/publicdomain/zero/1.0/) ) applies to the data made available in this article, unless otherwise stated in a credit line to the data.
spellingShingle Correction
Xu, Yun
Nasri, Masoud
Dannenmann, Benjamin
Mir, Perihan
Zahabi, Azadeh
Welte, Karl
Morishima, Tatsuya
Skokowa, Julia
Correction: NAMPT/SIRT2-mediated inhibition of the p53-p21 signaling pathway is indispensable for maintenance and hematopoietic differentiation of human iPS cells
title Correction: NAMPT/SIRT2-mediated inhibition of the p53-p21 signaling pathway is indispensable for maintenance and hematopoietic differentiation of human iPS cells
title_full Correction: NAMPT/SIRT2-mediated inhibition of the p53-p21 signaling pathway is indispensable for maintenance and hematopoietic differentiation of human iPS cells
title_fullStr Correction: NAMPT/SIRT2-mediated inhibition of the p53-p21 signaling pathway is indispensable for maintenance and hematopoietic differentiation of human iPS cells
title_full_unstemmed Correction: NAMPT/SIRT2-mediated inhibition of the p53-p21 signaling pathway is indispensable for maintenance and hematopoietic differentiation of human iPS cells
title_short Correction: NAMPT/SIRT2-mediated inhibition of the p53-p21 signaling pathway is indispensable for maintenance and hematopoietic differentiation of human iPS cells
title_sort correction: nampt/sirt2-mediated inhibition of the p53-p21 signaling pathway is indispensable for maintenance and hematopoietic differentiation of human ips cells
topic Correction
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC10588213/
https://www.ncbi.nlm.nih.gov/pubmed/37858174
http://dx.doi.org/10.1186/s13287-023-03511-4
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