Cargando…
JTE-013 Alleviates Pulmonary Fibrosis by Affecting the RhoA/YAP Pathway and Mitochondrial Fusion/Fission
Pulmonary fibrosis may be due to the proliferation of fibroblasts and the aggregation of extracellular matrix, resulting in the stimulation of inflammation damage, destroying lung tissue structure, seriously affecting the patient’s respiratory function, and even leading to death. We investigated the...
Autores principales: | Zhou, Jiaxu, Song, Yilan, Wang, Xingmei, Li, Xinrui, Liu, Chang, Tian, Chenchen, Wang, Chongyang, Li, Liangchang, Yan, Guanghai, Cui, Hong |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
MDPI
2023
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC10609863/ https://www.ncbi.nlm.nih.gov/pubmed/37895915 http://dx.doi.org/10.3390/ph16101444 |
Ejemplares similares
-
Administration of JTE013 abrogates experimental asthma by regulating proinflammatory cytokine production from bronchial epithelial cells
por: Terashita, Tomomi, et al.
Publicado: (2016) -
Inhibition of Sphingosine-1-Phosphate Receptor 2 by JTE013 Enhanced Alveolar Bone Regeneration by Promoting Angiogenesis
por: Lory, William, et al.
Publicado: (2023) -
The sphingosine 1-phosphate receptor 2/4 antagonist JTE-013 elicits off-target effects on sphingolipid metabolism
por: Pitman, Melissa R., et al.
Publicado: (2022) -
Topical Application of S1P(2) Antagonist JTE-013 Attenuates 2,4-Dinitrochlorobenzene-Induced Atopic Dermatitis in Mice
por: Kang, Jisoo, et al.
Publicado: (2020) -
Step-Double-Zone-JTE for SiC Devices with Increased Tolerance to JTE Dose and Surface Charges
por: Huang, Yifei, et al.
Publicado: (2018)