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Intrauterine Fetal Demise: A Rare Complication of Wernicke's Encephalopathy Secondary to Hyperemesis Gravidarum

Wernicke's encephalopathy (WE) is an acute neurological disorder caused by severe thiamine deficiency that manifests with a common range of clinical features including a triad of global confusion state, ophthalmoplegia, and ataxia. Though frequently associated with the alcohol-dependent populat...

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Autores principales: Pham, Anthony, Okpara, Robin, Rollins, Nancy, Jacob, Roy
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Cureus 2023
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC10655897/
https://www.ncbi.nlm.nih.gov/pubmed/38021939
http://dx.doi.org/10.7759/cureus.47270
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author Pham, Anthony
Okpara, Robin
Rollins, Nancy
Jacob, Roy
author_facet Pham, Anthony
Okpara, Robin
Rollins, Nancy
Jacob, Roy
author_sort Pham, Anthony
collection PubMed
description Wernicke's encephalopathy (WE) is an acute neurological disorder caused by severe thiamine deficiency that manifests with a common range of clinical features including a triad of global confusion state, ophthalmoplegia, and ataxia. Though frequently associated with the alcohol-dependent population, WE has been seen in other patients where it often goes undiagnosed presumably due to rarity and variable clinical indications. In this case report, we highlight the importance of WE being considered as a differential diagnosis of acute encephalopathy particularly in women who have experienced fetal demise in conjunction with signs of malnourishment from hyperemesis gravidarum.
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spelling pubmed-106558972023-10-18 Intrauterine Fetal Demise: A Rare Complication of Wernicke's Encephalopathy Secondary to Hyperemesis Gravidarum Pham, Anthony Okpara, Robin Rollins, Nancy Jacob, Roy Cureus Internal Medicine Wernicke's encephalopathy (WE) is an acute neurological disorder caused by severe thiamine deficiency that manifests with a common range of clinical features including a triad of global confusion state, ophthalmoplegia, and ataxia. Though frequently associated with the alcohol-dependent population, WE has been seen in other patients where it often goes undiagnosed presumably due to rarity and variable clinical indications. In this case report, we highlight the importance of WE being considered as a differential diagnosis of acute encephalopathy particularly in women who have experienced fetal demise in conjunction with signs of malnourishment from hyperemesis gravidarum. Cureus 2023-10-18 /pmc/articles/PMC10655897/ /pubmed/38021939 http://dx.doi.org/10.7759/cureus.47270 Text en Copyright © 2023, Pham et al. https://creativecommons.org/licenses/by/3.0/This is an open access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.
spellingShingle Internal Medicine
Pham, Anthony
Okpara, Robin
Rollins, Nancy
Jacob, Roy
Intrauterine Fetal Demise: A Rare Complication of Wernicke's Encephalopathy Secondary to Hyperemesis Gravidarum
title Intrauterine Fetal Demise: A Rare Complication of Wernicke's Encephalopathy Secondary to Hyperemesis Gravidarum
title_full Intrauterine Fetal Demise: A Rare Complication of Wernicke's Encephalopathy Secondary to Hyperemesis Gravidarum
title_fullStr Intrauterine Fetal Demise: A Rare Complication of Wernicke's Encephalopathy Secondary to Hyperemesis Gravidarum
title_full_unstemmed Intrauterine Fetal Demise: A Rare Complication of Wernicke's Encephalopathy Secondary to Hyperemesis Gravidarum
title_short Intrauterine Fetal Demise: A Rare Complication of Wernicke's Encephalopathy Secondary to Hyperemesis Gravidarum
title_sort intrauterine fetal demise: a rare complication of wernicke's encephalopathy secondary to hyperemesis gravidarum
topic Internal Medicine
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC10655897/
https://www.ncbi.nlm.nih.gov/pubmed/38021939
http://dx.doi.org/10.7759/cureus.47270
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