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Molecular Linkage between Immune System Disorders and Atherosclerosis

A strong relationship exists between immune dysfunction and cardiovascular disease. Immune dysregulation can promote the development of cardiovascular diseases as well as exacerbate their course. The disorders may occur due to the presence of primary immune defects (currently known as inborn errors...

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Autores principales: Napiórkowska-Baran, Katarzyna, Schmidt, Oskar, Szymczak, Bartłomiej, Lubański, Jakub, Doligalska, Agata, Bartuzi, Zbigniew
Formato: Online Artículo Texto
Lenguaje:English
Publicado: MDPI 2023
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC10670175/
https://www.ncbi.nlm.nih.gov/pubmed/37998729
http://dx.doi.org/10.3390/cimb45110552
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author Napiórkowska-Baran, Katarzyna
Schmidt, Oskar
Szymczak, Bartłomiej
Lubański, Jakub
Doligalska, Agata
Bartuzi, Zbigniew
author_facet Napiórkowska-Baran, Katarzyna
Schmidt, Oskar
Szymczak, Bartłomiej
Lubański, Jakub
Doligalska, Agata
Bartuzi, Zbigniew
author_sort Napiórkowska-Baran, Katarzyna
collection PubMed
description A strong relationship exists between immune dysfunction and cardiovascular disease. Immune dysregulation can promote the development of cardiovascular diseases as well as exacerbate their course. The disorders may occur due to the presence of primary immune defects (currently known as inborn errors of immunity) and the more common secondary immune deficiencies. Secondary immune deficiencies can be caused by certain chronic conditions (such as diabetes, chronic kidney disease, obesity, autoimmune diseases, or cancer), nutritional deficiencies (including both lack of nutrients and bioactive non-nutrient compounds), and medical treatments and addictive substances. This article unravels the molecular linkage between the aforementioned immune system disorders and atherosclerosis.
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spelling pubmed-106701752023-11-01 Molecular Linkage between Immune System Disorders and Atherosclerosis Napiórkowska-Baran, Katarzyna Schmidt, Oskar Szymczak, Bartłomiej Lubański, Jakub Doligalska, Agata Bartuzi, Zbigniew Curr Issues Mol Biol Review A strong relationship exists between immune dysfunction and cardiovascular disease. Immune dysregulation can promote the development of cardiovascular diseases as well as exacerbate their course. The disorders may occur due to the presence of primary immune defects (currently known as inborn errors of immunity) and the more common secondary immune deficiencies. Secondary immune deficiencies can be caused by certain chronic conditions (such as diabetes, chronic kidney disease, obesity, autoimmune diseases, or cancer), nutritional deficiencies (including both lack of nutrients and bioactive non-nutrient compounds), and medical treatments and addictive substances. This article unravels the molecular linkage between the aforementioned immune system disorders and atherosclerosis. MDPI 2023-11-01 /pmc/articles/PMC10670175/ /pubmed/37998729 http://dx.doi.org/10.3390/cimb45110552 Text en © 2023 by the authors. https://creativecommons.org/licenses/by/4.0/Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/).
spellingShingle Review
Napiórkowska-Baran, Katarzyna
Schmidt, Oskar
Szymczak, Bartłomiej
Lubański, Jakub
Doligalska, Agata
Bartuzi, Zbigniew
Molecular Linkage between Immune System Disorders and Atherosclerosis
title Molecular Linkage between Immune System Disorders and Atherosclerosis
title_full Molecular Linkage between Immune System Disorders and Atherosclerosis
title_fullStr Molecular Linkage between Immune System Disorders and Atherosclerosis
title_full_unstemmed Molecular Linkage between Immune System Disorders and Atherosclerosis
title_short Molecular Linkage between Immune System Disorders and Atherosclerosis
title_sort molecular linkage between immune system disorders and atherosclerosis
topic Review
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC10670175/
https://www.ncbi.nlm.nih.gov/pubmed/37998729
http://dx.doi.org/10.3390/cimb45110552
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