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E1A expression dysregulates IL-8 production and suppresses IL-6 production by lung epithelial cells
BACKGROUND: The adenoviral protein E1A has been proposed to play a role in the pathophysiology of COPD, in particular by increasing IL-8 gene transcription of lung epithelial cells in response to cigarette smoke-constituents such as LPS. As IL-8 production is also under tight post-transcriptional co...
| Autores principales: | , , , |
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| Formato: | Texto |
| Lenguaje: | English |
| Publicado: |
BioMed Central
2005
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| Materias: | |
| Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC1261537/ https://www.ncbi.nlm.nih.gov/pubmed/16185356 http://dx.doi.org/10.1186/1465-9921-6-111 |