Cargando…
Control of G1 arrest after DNA damage.
The temporal relationship between DNA damage and DNA replication may be critical in determining whether the genetic changes necessary for cellular transformation occur after DNA damage. Recent characterization of the mechanisms responsible for alterations in cell-cycle progression after DNA damage i...
Autores principales: | Kastan, M B, Kuerbitz, S J |
---|---|
Formato: | Texto |
Lenguaje: | English |
Publicado: |
1993
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC1519427/ https://www.ncbi.nlm.nih.gov/pubmed/8013425 |
Ejemplares similares
-
DNA damage response pathways in cancer causation and treatment
por: Kastan, MB, et al.
Publicado: (2005) -
A novel DNA damage-induced alternative splicing pathway that regulates p53 and cellular senescence markers
por: Chen, Jing, et al.
Publicado: (2017) -
DNA-Damage-Induced Alternative Splicing of p53
por: Chen, Jing, et al.
Publicado: (2021) -
Sustained CHK2 activity, but not ATM activity, is critical to maintain a G1 arrest after DNA damage in untransformed cells
por: García-Santisteban, Iraia, et al.
Publicado: (2021) -
Combined experimental and computational analysis of DNA damage signaling reveals context-dependent roles for Erk in apoptosis and G1/S arrest after genotoxic stress
por: Tentner, Andrea R, et al.
Publicado: (2012)