Reduced neuronal cell death after experimental brain injury in mice lacking a functional alternative pathway of complement activation

BACKGROUND: Neuroprotective strategies for prevention of the neuropathological sequelae of traumatic brain injury (TBI) have largely failed in translation to clinical treatment. Thus, there is a substantial need for further understanding the molecular mechanisms and pathways which lead to secondary...

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Detalles Bibliográficos
Autores principales: Leinhase, Iris, Holers, V Michael, Thurman, Joshua M, Harhausen, Denise, Schmidt, Oliver I, Pietzcker, Malte, Taha, Mohy E, Rittirsch, Daniel, Huber-Lang, Markus, Smith, Wade R, Ward, Peter A, Stahel, Philip F
Formato: Texto
Lenguaje:English
Publicado: BioMed Central 2006
Materias:
Research Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC1540436/
https://www.ncbi.nlm.nih.gov/pubmed/16842619
http://dx.doi.org/10.1186/1471-2202-7-55

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC1540436/
https://www.ncbi.nlm.nih.gov/pubmed/16842619
http://dx.doi.org/10.1186/1471-2202-7-55

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