Cargando…
Molecular mechanisms of transformation of C3H/10T1/2 C1 8 mouse embryo cells and diploid human fibroblasts by carcinogenic metal compounds.
Carcinogenic arsenic, nickel, and chromium compounds induced morphological and neoplastic transformation but no mutation to ouabain resistance in 10T1/2 mouse embryo cells; lead chromate also did not induce mutation to ouabain or 6-thioguanine resistance in Chinese hamster ovary cells. The mechanism...
Autor principal: | Landolph, J R |
---|---|
Formato: | Texto |
Lenguaje: | English |
Publicado: |
1994
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC1567381/ https://www.ncbi.nlm.nih.gov/pubmed/7843085 |
Ejemplares similares
-
Molecular biology of deregulated gene expression in transformed C3H/10T1/2 mouse embryo cell lines induced by specific insoluble carcinogenic nickel compounds.
por: Landolph, Joseph R, et al.
Publicado: (2002) -
Cytotoxic and mutagenic effects of specific carcinogen-DNA adducts in diploid human fibroblasts.
por: McCormick, J J, et al.
Publicado: (1985) -
Insoluble Ni compound-induced gene amplification/gene silencing causes over-expression of microtubules/microfilaments, cell shape changes, and de-regulation of global gene expression/Ca+(2) gradients, inducing morphological/ neoplastic transformation of 10T1/2 mouse embryo cells
por: Landolph, Joseph, et al.
Publicado: (2010) -
Activation of cellular oncogenes by chemical carcinogens in Syrian hamster embryo fibroblasts.
por: Ebert, R, et al.
Publicado: (1990) -
Biomonitoring exposure to metal compounds with carcinogenic properties.
por: Léonard, A, et al.
Publicado: (1993)