Cargando…
Release of tumor necrosis factor-alpha and prostanoids in whole blood cultures after in vivo exposure to low-dose aspirin.
BACKGROUND: The preventive effect of low-dose aspirin in cardiovascular disease is generally attributed to its antiplatelet action caused by differential inhibition of platelet cyclooxygenase-1. However, there is evidence that aspirin also affects release of inflammatory cytokines, including tumor n...
Autores principales: | Beckmann, I, Ben-Efraim, S, Vervoort, M, Wallenburg, H C |
---|---|
Formato: | Texto |
Lenguaje: | English |
Publicado: |
2001
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC1781693/ https://www.ncbi.nlm.nih.gov/pubmed/11405554 |
Ejemplares similares
-
Physiology and pathophysiology of prostanoid receptors
por: Narumiya, Shuh
Publicado: (2007) -
Prostanoid Receptors in the Human Vascular Wall
por: Norel, Xavier
Publicado: (2007) -
Prostanoid Metabolites as Biomarkers in Human Disease
por: Idborg, Helena, et al.
Publicado: (2022) -
The Link between Prostanoids and Cardiovascular Diseases
por: Beccacece, Livia, et al.
Publicado: (2023) -
Imbalanced prostanoid release mediates cigarette smoke-induced human pulmonary artery cell proliferation
por: Alqarni, Abdullah A., et al.
Publicado: (2022)