Cargando…
Dynorphin Activates Quorum Sensing Quinolone Signaling in Pseudomonas aeruginosa
There is now substantial evidence that compounds released during host stress directly activate the virulence of certain opportunistic pathogens. Here, we considered that endogenous opioids might function as such compounds, given that they are among the first signals to be released at multiple tissue...
Autores principales: | Zaborina, Olga, Lepine, Francois, Xiao, Gaoping, Valuckaite, Vesta, Chen, Yimei, Li, Terry, Ciancio, Mae, Zaborin, Alex, Petroff, Elaine, Turner, Jerrold R, Rahme, Laurence G, Chang, Eugene, Alverdy, John C |
---|---|
Formato: | Texto |
Lenguaje: | English |
Publicado: |
Public Library of Science
2007
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC1828698/ https://www.ncbi.nlm.nih.gov/pubmed/17367209 http://dx.doi.org/10.1371/journal.ppat.0030035 |
Ejemplares similares
-
Correction: Dynorphin Activates Quorum Sensing Quinolone Signaling in Pseudomonas aeruginosa
por: Zaborina, Olga, et al.
Publicado: (2007) -
Prevention of siderophore- mediated gut-derived sepsis due to P. aeruginosa can be achieved without iron provision by maintaining local phosphate abundance: role of pH
por: Romanowski, Kathleen, et al.
Publicado: (2011) -
Intestinal Tissues Induce an SNP Mutation in Pseudomonas aeruginosa That Enhances Its Virulence: Possible Role in Anastomotic Leak
por: Olivas, Andrea D., et al.
Publicado: (2012) -
Structure–Function Aspects of PstS in Multi-Drug–Resistant Pseudomonas aeruginosa
por: Zaborina, Olga, et al.
Publicado: (2008) -
Candida albicans Isolates from the Gut of Critically Ill Patients Respond to Phosphate Limitation by Expressing Filaments and a Lethal Phenotype
por: Romanowski, Kathleen, et al.
Publicado: (2012)