Cargando…
Deletion of Fcγ Receptor IIB Renders H-2(b) Mice Susceptible to Collagen-induced Arthritis
Autoimmune diseases, like rheumatoid arthritis, result from a dysregulation of the immune response culminating in hyperactivation of effector cells leading to immune-mediated injury. To maintain an appropriate immune response and prevent the emergence of autoimmune disease, activation signals must b...
Autores principales: | Yuasa, Takae, Kubo, Satoshi, Yoshino, Tadashi, Ujike, Azusa, Matsumura, Kimio, Ono, Masao, Ravetch, Jeffrey V., Takai, Toshiyuki |
---|---|
Formato: | Texto |
Lenguaje: | English |
Publicado: |
The Rockefeller University Press
1999
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC1887699/ https://www.ncbi.nlm.nih.gov/pubmed/9874575 |
Ejemplares similares
-
Fcγ Receptor Iib–Deficient Mice Develop Goodpasture's Syndrome upon Immunization with Type IV Collagen: A Novel Murine Model for Autoimmune Glomerular Basement Membrane Disease
por: Nakamura, Akira, et al.
Publicado: (2000) -
Modulation of Immunoglobulin (Ig)E-mediated Systemic Anaphylaxis by Low-Affinity Fc Receptors for IgG
por: Ujike, Azusa, et al.
Publicado: (1999) -
Lyn Is Essential for Fcγ Receptor III–Mediated Systemic Anaphylaxis but Not for the Arthus Reaction
por: Yuasa, Takae, et al.
Publicado: (2001) -
Selective dysregulation of the FcγIIB receptor on memory B cells in SLE
por: Mackay, Meggan, et al.
Publicado: (2006) -
Modulation of Immune Complex–induced Inflammation In Vivo by the Coordinate Expression of Activation and Inhibitory Fc Receptors
por: Clynes, Raphael, et al.
Publicado: (1999)