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Role of the calmodulin inhibitor trifluoperazine on the induction and expression of cell cycle traverse perturbations and cytotoxicity of daunorubicin and doxorubicin (adriamycin) in doxorubicin-resistant P388 mouse leukaemia cells.

Detalles Bibliográficos
Autores principales: Ganapathi, R., Yen, A., Grabowski, D., Schmidt, H., Turinic, R., Valenzuela, R.
Formato: Texto
Lenguaje:English
Publicado: Nature Publishing Group 1986
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2001449/
https://www.ncbi.nlm.nih.gov/pubmed/3707847
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author Ganapathi, R.
Yen, A.
Grabowski, D.
Schmidt, H.
Turinic, R.
Valenzuela, R.
author_facet Ganapathi, R.
Yen, A.
Grabowski, D.
Schmidt, H.
Turinic, R.
Valenzuela, R.
author_sort Ganapathi, R.
collection PubMed
description
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institution National Center for Biotechnology Information
language English
publishDate 1986
publisher Nature Publishing Group
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spelling pubmed-20014492009-09-10 Role of the calmodulin inhibitor trifluoperazine on the induction and expression of cell cycle traverse perturbations and cytotoxicity of daunorubicin and doxorubicin (adriamycin) in doxorubicin-resistant P388 mouse leukaemia cells. Ganapathi, R. Yen, A. Grabowski, D. Schmidt, H. Turinic, R. Valenzuela, R. Br J Cancer Research Article Nature Publishing Group 1986-04 /pmc/articles/PMC2001449/ /pubmed/3707847 Text en https://creativecommons.org/licenses/by/4.0/This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit https://creativecommons.org/licenses/by/4.0/.
spellingShingle Research Article
Ganapathi, R.
Yen, A.
Grabowski, D.
Schmidt, H.
Turinic, R.
Valenzuela, R.
Role of the calmodulin inhibitor trifluoperazine on the induction and expression of cell cycle traverse perturbations and cytotoxicity of daunorubicin and doxorubicin (adriamycin) in doxorubicin-resistant P388 mouse leukaemia cells.
title Role of the calmodulin inhibitor trifluoperazine on the induction and expression of cell cycle traverse perturbations and cytotoxicity of daunorubicin and doxorubicin (adriamycin) in doxorubicin-resistant P388 mouse leukaemia cells.
title_full Role of the calmodulin inhibitor trifluoperazine on the induction and expression of cell cycle traverse perturbations and cytotoxicity of daunorubicin and doxorubicin (adriamycin) in doxorubicin-resistant P388 mouse leukaemia cells.
title_fullStr Role of the calmodulin inhibitor trifluoperazine on the induction and expression of cell cycle traverse perturbations and cytotoxicity of daunorubicin and doxorubicin (adriamycin) in doxorubicin-resistant P388 mouse leukaemia cells.
title_full_unstemmed Role of the calmodulin inhibitor trifluoperazine on the induction and expression of cell cycle traverse perturbations and cytotoxicity of daunorubicin and doxorubicin (adriamycin) in doxorubicin-resistant P388 mouse leukaemia cells.
title_short Role of the calmodulin inhibitor trifluoperazine on the induction and expression of cell cycle traverse perturbations and cytotoxicity of daunorubicin and doxorubicin (adriamycin) in doxorubicin-resistant P388 mouse leukaemia cells.
title_sort role of the calmodulin inhibitor trifluoperazine on the induction and expression of cell cycle traverse perturbations and cytotoxicity of daunorubicin and doxorubicin (adriamycin) in doxorubicin-resistant p388 mouse leukaemia cells.
topic Research Article
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2001449/
https://www.ncbi.nlm.nih.gov/pubmed/3707847
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