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The innate resistance of CBA mice to endogenous murine leukaemia virus infection.
The incidence of lymphomata in CBA mice is low and furthermore is unaltered by transplantation at the early blastocyst stage and being born from the lymphoma-prone AKR. The number of C-type murine leukaemia virus particles in CBA derived in this manner and milk-fostered by AKR mice in no way differs...
Autores principales: | , , , , |
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Formato: | Texto |
Lenguaje: | English |
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Nature Publishing Group
1976
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Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2025133/ https://www.ncbi.nlm.nih.gov/pubmed/182192 |
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author | Barnes, R. D. Tuffrey, M. Willis, E. J. Mahouy, G. Lasneret, J. |
author_facet | Barnes, R. D. Tuffrey, M. Willis, E. J. Mahouy, G. Lasneret, J. |
author_sort | Barnes, R. D. |
collection | PubMed |
description | The incidence of lymphomata in CBA mice is low and furthermore is unaltered by transplantation at the early blastocyst stage and being born from the lymphoma-prone AKR. The number of C-type murine leukaemia virus particles in CBA derived in this manner and milk-fostered by AKR mice in no way differs from normal CBA. The results suggest that the oncogenic Gross virus does not pass through either the transplacental or transmammary routes, or alternatively that viral replication in the CBA was in some way inhibited. Both possibilities have still to be distinguished. IMAGES: |
format | Text |
id | pubmed-2025133 |
institution | National Center for Biotechnology Information |
language | English |
publishDate | 1976 |
publisher | Nature Publishing Group |
record_format | MEDLINE/PubMed |
spelling | pubmed-20251332009-09-10 The innate resistance of CBA mice to endogenous murine leukaemia virus infection. Barnes, R. D. Tuffrey, M. Willis, E. J. Mahouy, G. Lasneret, J. Br J Cancer Research Article The incidence of lymphomata in CBA mice is low and furthermore is unaltered by transplantation at the early blastocyst stage and being born from the lymphoma-prone AKR. The number of C-type murine leukaemia virus particles in CBA derived in this manner and milk-fostered by AKR mice in no way differs from normal CBA. The results suggest that the oncogenic Gross virus does not pass through either the transplacental or transmammary routes, or alternatively that viral replication in the CBA was in some way inhibited. Both possibilities have still to be distinguished. IMAGES: Nature Publishing Group 1976-07 /pmc/articles/PMC2025133/ /pubmed/182192 Text en https://creativecommons.org/licenses/by/4.0/This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit https://creativecommons.org/licenses/by/4.0/. |
spellingShingle | Research Article Barnes, R. D. Tuffrey, M. Willis, E. J. Mahouy, G. Lasneret, J. The innate resistance of CBA mice to endogenous murine leukaemia virus infection. |
title | The innate resistance of CBA mice to endogenous murine leukaemia virus infection. |
title_full | The innate resistance of CBA mice to endogenous murine leukaemia virus infection. |
title_fullStr | The innate resistance of CBA mice to endogenous murine leukaemia virus infection. |
title_full_unstemmed | The innate resistance of CBA mice to endogenous murine leukaemia virus infection. |
title_short | The innate resistance of CBA mice to endogenous murine leukaemia virus infection. |
title_sort | innate resistance of cba mice to endogenous murine leukaemia virus infection. |
topic | Research Article |
url | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2025133/ https://www.ncbi.nlm.nih.gov/pubmed/182192 |
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