Cargando…
Adenosine A(2A) receptor activation reduces hepatic ischemia reperfusion injury by inhibiting CD1d-dependent NKT cell activation
Ischemia reperfusion injury results from tissue damage during ischemia and ongoing inflammation and injury during reperfusion. Liver reperfusion injury is reduced by lymphocyte depletion or activation of adenosine A(2A) receptors (A(2A)Rs) with the selective agonist 4- {3-[6-amino-9-(5-ethylcarbamoy...
Autores principales: | Lappas, Courtney M., Day, Yuan-Ji, Marshall, Melissa A., Engelhard, Victor H., Linden, Joel |
---|---|
Formato: | Texto |
Lenguaje: | English |
Publicado: |
The Rockefeller University Press
2006
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2118143/ https://www.ncbi.nlm.nih.gov/pubmed/17088433 http://dx.doi.org/10.1084/jem.20061097 |
Ejemplares similares
-
Protection from pulmonary ischemia-reperfusion injury by adenosine A2A receptor activation
por: Sharma, Ashish K, et al.
Publicado: (2009) -
NKT cells are important mediators of hepatic ischemia-reperfusion injury
por: Richards, James A., et al.
Publicado: (2017) -
Activation of iNKT Cells Facilitates Liver Repair After Hepatic Ischemia Reperfusion Injury Through Acceleration of Macrophage Polarization
por: Goto, Takuya, et al.
Publicado: (2021) -
NF-κB Is Activated in CD4(+) iNKT Cells by Sickle Cell Disease and Mediates Rapid Induction of Adenosine A(2A) Receptors
por: Lin, Gene, et al.
Publicado: (2013) -
Rapamycin protects kidney against ischemia reperfusion injury through recruitment of NKT cells
por: Zhang, Chao, et al.
Publicado: (2014)