THE INCREASED SUSCEPTIBILITY TO HEMOLYSIS BY INDOL IN DOGS FED DEFICIENT DIETS

1. Indol is more hemolytic in the presence of a deficiency complex than when a normal diet is fed. 2. The hemolytic effect can be abolished by supplementing the deficient diet with liver extract curative of pernicious anemia in man. 3. The hemolysis affects all hemoglobin-containing cells, including...

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Detalles Bibliográficos
Autores principales: Rhoads, C. P., Barker, W. Halsey, Miller, D. K.
Formato: Texto
Lenguaje:English
Publicado: The Rockefeller University Press 1938
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2133558/
https://www.ncbi.nlm.nih.gov/pubmed/19870721
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author Rhoads, C. P.
Barker, W. Halsey
Miller, D. K.
author_facet Rhoads, C. P.
Barker, W. Halsey
Miller, D. K.
author_sort Rhoads, C. P.
collection PubMed
description 1. Indol is more hemolytic in the presence of a deficiency complex than when a normal diet is fed. 2. The hemolytic effect can be abolished by supplementing the deficient diet with liver extract curative of pernicious anemia in man. 3. The hemolysis affects all hemoglobin-containing cells, including reticulocytes. 4. The repair of the anemia resulting from the administration of indol in the presence of a deficiency represents the cessation of a hemolytic process. 5. An abnormally low rate of production of erythrocytes may well be a factor in the production of the anemia.
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spelling pubmed-21335582008-04-18 THE INCREASED SUSCEPTIBILITY TO HEMOLYSIS BY INDOL IN DOGS FED DEFICIENT DIETS Rhoads, C. P. Barker, W. Halsey Miller, D. K. J Exp Med Article 1. Indol is more hemolytic in the presence of a deficiency complex than when a normal diet is fed. 2. The hemolytic effect can be abolished by supplementing the deficient diet with liver extract curative of pernicious anemia in man. 3. The hemolysis affects all hemoglobin-containing cells, including reticulocytes. 4. The repair of the anemia resulting from the administration of indol in the presence of a deficiency represents the cessation of a hemolytic process. 5. An abnormally low rate of production of erythrocytes may well be a factor in the production of the anemia. The Rockefeller University Press 1938-01-31 /pmc/articles/PMC2133558/ /pubmed/19870721 Text en Copyright © Copyright, 1938, by The Rockefeller Institute for Medical Research New York This article is distributed under the terms of an Attribution–Noncommercial–Share Alike–No Mirror Sites license for the first six months after the publication date (see http://www.rupress.org/terms). After six months it is available under a Creative Commons License (Attribution–Noncommercial–Share Alike 4.0 Unported license, as described at http://creativecommons.org/licenses/by-nc-sa/4.0/).
spellingShingle Article
Rhoads, C. P.
Barker, W. Halsey
Miller, D. K.
THE INCREASED SUSCEPTIBILITY TO HEMOLYSIS BY INDOL IN DOGS FED DEFICIENT DIETS
title THE INCREASED SUSCEPTIBILITY TO HEMOLYSIS BY INDOL IN DOGS FED DEFICIENT DIETS
title_full THE INCREASED SUSCEPTIBILITY TO HEMOLYSIS BY INDOL IN DOGS FED DEFICIENT DIETS
title_fullStr THE INCREASED SUSCEPTIBILITY TO HEMOLYSIS BY INDOL IN DOGS FED DEFICIENT DIETS
title_full_unstemmed THE INCREASED SUSCEPTIBILITY TO HEMOLYSIS BY INDOL IN DOGS FED DEFICIENT DIETS
title_short THE INCREASED SUSCEPTIBILITY TO HEMOLYSIS BY INDOL IN DOGS FED DEFICIENT DIETS
title_sort increased susceptibility to hemolysis by indol in dogs fed deficient diets
topic Article
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2133558/
https://www.ncbi.nlm.nih.gov/pubmed/19870721
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