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INJURY IN RELATION TO CELL ORGANIZATION
When a part of a Nitella cell, A, is covered with water and the rest of the cell, B, is in contact with a toxic solution there is an escape of solutes at B. This is followed by the escape of solutes at A which causes the death of A. Water enters at A, flows along inside the cell, and escapes at B ca...
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Formato: | Texto |
Lenguaje: | English |
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The Rockefeller University Press
1951
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Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2147223/ https://www.ncbi.nlm.nih.gov/pubmed/14824500 |
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author | Osterhout, W. J. V. |
author_facet | Osterhout, W. J. V. |
author_sort | Osterhout, W. J. V. |
collection | PubMed |
description | When a part of a Nitella cell, A, is covered with water and the rest of the cell, B, is in contact with a toxic solution there is an escape of solutes at B. This is followed by the escape of solutes at A which causes the death of A. Water enters at A, flows along inside the cell, and escapes at B carrying solutes with it. When this is prevented by covering A with mineral oil the escape of solutes at A is delayed and the life of A is correspondingly prolonged. It is remarkable that this occurs in spite of the fact that the hydrostatic pressure inside the cell (turgor) drops from 6.4 atmospheres to zero. It would seem that A might not be affected by the death of B if the escape of solutes could be prevented. |
format | Text |
id | pubmed-2147223 |
institution | National Center for Biotechnology Information |
language | English |
publishDate | 1951 |
publisher | The Rockefeller University Press |
record_format | MEDLINE/PubMed |
spelling | pubmed-21472232008-04-23 INJURY IN RELATION TO CELL ORGANIZATION Osterhout, W. J. V. J Gen Physiol Article When a part of a Nitella cell, A, is covered with water and the rest of the cell, B, is in contact with a toxic solution there is an escape of solutes at B. This is followed by the escape of solutes at A which causes the death of A. Water enters at A, flows along inside the cell, and escapes at B carrying solutes with it. When this is prevented by covering A with mineral oil the escape of solutes at A is delayed and the life of A is correspondingly prolonged. It is remarkable that this occurs in spite of the fact that the hydrostatic pressure inside the cell (turgor) drops from 6.4 atmospheres to zero. It would seem that A might not be affected by the death of B if the escape of solutes could be prevented. The Rockefeller University Press 1951-01-20 /pmc/articles/PMC2147223/ /pubmed/14824500 Text en Copyright © Copyright, 1951, by The Rockefeller Institute for Medical Research This article is distributed under the terms of an Attribution–Noncommercial–Share Alike–No Mirror Sites license for the first six months after the publication date (see http://www.rupress.org/terms). After six months it is available under a Creative Commons License (Attribution–Noncommercial–Share Alike 4.0 Unported license, as described at http://creativecommons.org/licenses/by-nc-sa/4.0/). |
spellingShingle | Article Osterhout, W. J. V. INJURY IN RELATION TO CELL ORGANIZATION |
title | INJURY IN RELATION TO CELL ORGANIZATION |
title_full | INJURY IN RELATION TO CELL ORGANIZATION |
title_fullStr | INJURY IN RELATION TO CELL ORGANIZATION |
title_full_unstemmed | INJURY IN RELATION TO CELL ORGANIZATION |
title_short | INJURY IN RELATION TO CELL ORGANIZATION |
title_sort | injury in relation to cell organization |
topic | Article |
url | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2147223/ https://www.ncbi.nlm.nih.gov/pubmed/14824500 |
work_keys_str_mv | AT osterhoutwjv injuryinrelationtocellorganization |