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A nitric oxide synthase transgene ameliorates muscular dystrophy in mdx mice
Dystrophin-deficient muscles experience large reductions in expression of nitric oxide synthase (NOS), which suggests that NO deficiency may influence the dystrophic pathology. Because NO can function as an antiinflammatory and cytoprotective molecule, we propose that the loss of NOS from dystrophic...
| Autores principales: | , , |
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| Formato: | Texto |
| Lenguaje: | English |
| Publicado: |
The Rockefeller University Press
2001
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| Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2150800/ https://www.ncbi.nlm.nih.gov/pubmed/11581289 http://dx.doi.org/10.1083/jcb.200105110 |