Cargando…
CEACAM engagement by human pathogens enhances cell adhesion and counteracts bacteria-induced detachment of epithelial cells
Exfoliation, which is the detachment of infected epithelial cells, is an innate defense mechanism to prevent bacterial colonization. Indeed, infection with Neisseria gonorrhoeae induced epithelial detachment from an extracellular matrix (ECM) substrate in vitro. Surprisingly, variants of N. gonorrho...
Autores principales: | Muenzner, Petra, Rohde, Manfred, Kneitz, Susanne, Hauck, Christof R. |
---|---|
Formato: | Texto |
Lenguaje: | English |
Publicado: |
The Rockefeller University Press
2005
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2171332/ https://www.ncbi.nlm.nih.gov/pubmed/16115956 http://dx.doi.org/10.1083/jcb.200412151 |
Ejemplares similares
-
Signaling by epithelial members of the CEACAM family – mucosal docking sites for pathogenic bacteria
por: Tchoupa, Arnaud Kengmo, et al.
Publicado: (2014) -
CEACAMs: their role in physiology and pathophysiology
por: Kuespert, Katharina, et al.
Publicado: (2006) -
CEACAM3—A Prim(at)e Invention for Opsonin-Independent Phagocytosis of Bacteria
por: Bonsignore, Patrizia, et al.
Publicado: (2020) -
CEACAM1 recognition by bacterial pathogens is species-specific
por: Voges, Maike, et al.
Publicado: (2010) -
Granulocyte CEACAM3 Is a Phagocytic Receptor of the Innate Immune System that Mediates Recognition and Elimination of Human-specific Pathogens
por: Schmitter, Tim, et al.
Publicado: (2004)